Constitutive Ret activity in knock-in multiple endocrine neoplasia type B mice induces profound elevation of brain dopamine concentration via enhanced synthesis and increases the number of TH-positive cells in the substantia nigra.
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GDNF Overexpression from the Native Locus Reveals its Role in the Nigrostriatal Dopaminergic System FunctionGDNF is not required for catecholaminergic neuron survival in vivoGDNF-Ret signaling in midbrain dopaminergic neurons and its implication for Parkinson diseasemRNA and microRNA analysis reveals modulation of biochemical pathways related to addiction in the ventral tegmental area of methamphetamine self-administering ratsNigral GFRα1 infusion in aged rats increases locomotor activity, nigral tyrosine hydroxylase, and dopamine content in synchronicity.RET PLCγ phosphotyrosine binding domain regulates Ca2+ signaling and neocortical neuronal migration.Altered gene expression profiles in the hippocampus and prefrontal cortex of type 2 diabetic ratsStructure and physiology of the RET receptor tyrosine kinase.Neurotrophic factor receptor RET: structure, cell biology, and inherited diseases.Exercise-Mediated Increase in Nigral Tyrosine Hydroxylase Is Accompanied by Increased Nigral GFR-α1 and EAAC1 Expression in Aging RatsDifferential effects of the dopamine neurotoxin MPTP in animals with a partial deletion of the GDNF receptor, GFR alpha1, geneRet rescues mitochondrial morphology and muscle degeneration of Drosophila Pink1 mutants.Nigral injection of a proteasomal inhibitor, lactacystin, induces widespread glial cell activation and shows various phenotypes of Parkinson's disease in young and adult mouse.Characterization of the striatal dopaminergic neurotransmission in MEN2B mice with elevated cerebral tissue dopamine.Removal of prolyl oligopeptidase reduces alpha-synuclein toxicity in cells and in vivo.N-Methyl, N-propynyl-2-phenylethylamine (MPPE), a Selegiline Analog, Attenuates MPTP-induced Dopaminergic Toxicity with Guaranteed Behavioral Safety: Involvement of Inhibitions of Mitochondrial Oxidative Burdens and p53 Gene-elicited Pro-apoptotic CConstitutive Ret signaling leads to long-lasting expression of amphetamine-induced place conditioning via elevation of mesolimbic dopamine.Manganese-Mediated Decrease in Levels of c-RET and Tyrosine Hydroxylase Expression In Vitro.Post-stroke Intranasal (+)-Naloxone Delivery Reduces Microglial Activation and Improves Behavioral Recovery from Ischemic Injury.
P2860
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P2860
Constitutive Ret activity in knock-in multiple endocrine neoplasia type B mice induces profound elevation of brain dopamine concentration via enhanced synthesis and increases the number of TH-positive cells in the substantia nigra.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Constitutive Ret activity in k ...... cells in the substantia nigra.
@en
type
label
Constitutive Ret activity in k ...... cells in the substantia nigra.
@en
prefLabel
Constitutive Ret activity in k ...... cells in the substantia nigra.
@en
P2093
P50
P1476
Constitutive Ret activity in k ...... cells in the substantia nigra.
@en
P2093
Atso Raasmaja
Frank Costantini
Jelena Mijatovic
Liisa Ahtee
Mart Saarma
Petri Auvinen
P304
P356
10.1523/JNEUROSCI.5647-06.2007
P407
P577
2007-05-01T00:00:00Z