Novel transcriptional regulation of the schlafen-2 gene in macrophages in response to TLR-triggered stimulation.
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Codon-usage-based inhibition of HIV protein synthesis by human schlafen 11To translate, or not to translate: viral and host mRNA regulation by interferon-stimulated genesThe P-loop region of Schlafen 3 acts within the cytosol to induce differentiation of human Caco-2 intestinal epithelial cells.Transcriptional signatures of Itk-deficient CD3+, CD4+ and CD8+ T-cellsSchlafen 1 inhibits the proliferation and tube formation of endothelial progenitor cellsRole of interferon {alpha} (IFN{alpha})-inducible Schlafen-5 in regulation of anchorage-independent growth and invasion of malignant melanoma cells.Identification of novel innate immune genes by transcriptional profiling of macrophages stimulated with TLR ligands.High SLFN11 expression predicts better survival for patients with KRAS exon 2 wild type colorectal cancer after treated with adjuvant oxaliplatin-based treatmentCancer, Warts, or Asymptomatic Infections: Clinical Presentation Matches Codon Usage Preferences in Human PapillomavirusesSchlafen 3, a novel gene, regulates colonic mucosal growth during aging.The unfolded protein response contributes to preimplantation mouse embryo death in the DDK syndrome.Expression and regulatory effects of murine Schlafen (Slfn) genes in malignant melanoma and renal cell carcinoma.Role of Schlafen 2 (SLFN2) in the generation of interferon alpha-induced growth inhibitory responsesCellular and Biochemical Mechanisms of the Retroviral Restriction Factor SAMHD1.Quieting T cells with Slfn2.The schlafen family of proteins and their regulation by interferons.Brucella melitensis, B. neotomae and B. ovis elicit common and distinctive macrophage defense transcriptional responsesThe Schlafen family: complex roles in different cell types and virus replication.From mammals to viruses: the Schlafen genes in developmental, proliferative and immune processes.Schlafen-3: a novel regulator of intestinal differentiation.Loss of T-cell quiescence by targeting Slfn2 prevents the development and progression of T-ALL.Expression and regulation of Schlafen (SLFN) family members in primary human monocytes, monocyte-derived dendritic cells and T cells.Combination of host susceptibility and Mycobacterium tuberculosis virulence define gene expression profile in the host.Slfn2 mutation-induced loss of T-cell quiescence leads to elevated de novo sterol synthesis.Schlafen2 mutation in mice causes an osteopetrotic phenotype due to a decrease in the number of osteoclast progenitors
P2860
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P2860
Novel transcriptional regulation of the schlafen-2 gene in macrophages in response to TLR-triggered stimulation.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Novel transcriptional regulati ...... to TLR-triggered stimulation.
@en
type
label
Novel transcriptional regulati ...... to TLR-triggered stimulation.
@en
prefLabel
Novel transcriptional regulati ...... to TLR-triggered stimulation.
@en
P2093
P1433
P1476
Novel transcriptional regulati ...... to TLR-triggered stimulation.
@en
P2093
Dongbum Kim
Doo-Sik Kim
Hyung-Joo Kwon
Keun-Wook Lee
Min-Soo Kim
Sanghoon Kwon
Wern-Joo Sohn
Younghee Lee
P304
P356
10.1016/J.MOLIMM.2007.03.001
P577
2007-04-16T00:00:00Z