Sustained induction of NF-kappa B is required for efficient expression of latent human immunodeficiency virus type 1.
about
Post-transcriptional gene silencing, transcriptional gene silencing and human immunodeficiency virusTranscriptional control of HIV latency: cellular signaling pathways, epigenetics, happenstance and the hope for a cureRecent developments in human immunodeficiency virus-1 latency researchStructural Basis of HIV-1 Activation by NF-κB—A Higher-Order Complex of p50:RelA Bound to the HIV-1 LTRThe roles of HIV-1 proteins and antiretroviral drug therapy in HIV-1-associated endothelial dysfunctionEpigenetic regulation of HIV-1 latency by cytosine methylationPhosphorylation of CDK9 at Ser175 enhances HIV transcription and is a marker of activated P-TEFb in CD4(+) T lymphocytesControl of stochastic gene expression by host factors at the HIV promoter.Yeast genetic analysis reveals the involvement of chromatin reassembly factors in repressing HIV-1 basal transcriptionCombinatorial signals from CD28 differentially regulate human immunodeficiency virus transcription in T cellsNegative elongation factor is required for the maintenance of proviral latency but does not induce promoter-proximal pausing of RNA polymerase II on the HIV long terminal repeat.Short communication: Methamphetamine treatment increases in vitro and in vivo HIV replication.Combinatorial latency reactivation for HIV-1 subtypes and variantsBET bromodomain inhibition as a novel strategy for reactivation of HIV-1Combination of biological screening in a cellular model of viral latency and virtual screening identifies novel compounds that reactivate HIV-1A flexible model of HIV-1 latency permitting evaluation of many primary CD4 T-cell reservoirs.Mycobacterium tuberculosis modulates the gene interactions to activate the HIV replication and faster disease progression in a co-infected hostHIV-1 reactivation induced by the periodontal pathogens Fusobacterium nucleatum and Porphyromonas gingivalis involves Toll-like receptor 2 [corrected] and 9 activation in monocytes/macrophages.Bovine foamy virus transactivator BTas interacts with cellular RelB to enhance viral transcriptionThe molecular biology of HIV latency: breaking and restoring the Tat-dependent transcriptional circuit.MiR-125b reduces porcine reproductive and respiratory syndrome virus replication by negatively regulating the NF-κB pathwayDendritic cell-induced activation of latent HIV-1 provirus in actively proliferating primary T lymphocytesInnate and adaptive factors regulating human immunodeficiency virus type 1 genomic activation.Caveolin 1 inhibits HIV replication by transcriptional repression mediated through NF-κB.Viral-host interactions that control HIV-1 transcriptional elongationCalcium/calcineurin synergizes with prostratin to promote NF-κB dependent activation of latent HIVThe oral commensal, Streptococcus gordonii, synergizes with Tat protein to induce HIV-1 promoter activation in monocytes/macrophagesHigh-throughput screening uncovers a compound that activates latent HIV-1 and acts cooperatively with a histone deacetylase (HDAC) inhibitor.T-cell receptor signaling enhances transcriptional elongation from latent HIV proviruses by activating P-TEFb through an ERK-dependent pathway.Epigenetic silencing of HIV-1 by the histone H3 lysine 27 methyltransferase enhancer of Zeste 2.Selective HDAC inhibition for the disruption of latent HIV-1 infection.Differential regulation of NF-κB-mediated proviral and antiviral host gene expression by primate lentiviral Nef and Vpu proteins.Identification of novel T cell factor 4 (TCF-4) binding sites on the HIV long terminal repeat which associate with TCF-4, β-catenin, and SMAR1 to repress HIV transcriptionTherapeutic doses of irradiation activate viral transcription and induce apoptosis in HIV-1 infected cells.Transcriptional burst frequency and burst size are equally modulated across the human genome.NF-κB/Rel: agonist and antagonist roles in HIV-1 latency.The histone deacetylase inhibitor ITF2357 decreases surface CXCR4 and CCR5 expression on CD4(+) T-cells and monocytes and is superior to valproic acid for latent HIV-1 expression in vitro.Interaction between lactobacilli, bacterial vaginosis-associated bacteria, and HIV Type 1 RNA and DNA Genital shedding in U.S. and Kenyan women.Preclinical evaluation of HIV eradication strategies in the simian immunodeficiency virus-infected rhesus macaque: a pilot study testing inhibition of indoleamine 2,3-dioxygenase.HSP70 binding protein 1 (HspBP1) suppresses HIV-1 replication by inhibiting NF-κB mediated activation of viral gene expression.
P2860
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P2860
Sustained induction of NF-kappa B is required for efficient expression of latent human immunodeficiency virus type 1.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Sustained induction of NF-kapp ...... immunodeficiency virus type 1.
@en
type
label
Sustained induction of NF-kapp ...... immunodeficiency virus type 1.
@en
prefLabel
Sustained induction of NF-kapp ...... immunodeficiency virus type 1.
@en
P2093
P2860
P356
P1433
P1476
Sustained induction of NF-kapp ...... immunodeficiency virus type 1.
@en
P2093
Hakju Kwon
Lin-Feng Chen
Samuel A Williams
Warner C Greene
P2860
P304
P356
10.1128/JVI.02074-06
P407
P577
2007-03-21T00:00:00Z