NF-kappaB activation by depolarization of skeletal muscle cells depends on ryanodine and IP3 receptor-mediated calcium signals.
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Lack of CFTR in skeletal muscle predisposes to muscle wasting and diaphragm muscle pump failure in cystic fibrosis miceChromogranin B regulates calcium signaling, nuclear factor kappaB activity, and brain natriuretic peptide production in cardiomyocytesCardiac-specific suppression of NF-κB signaling prevents diabetic cardiomyopathy via inhibition of the renin-angiotensin system.Lipopolysaccharide induces endoplasmic store Ca2+-dependent inflammatory responses in lung microvesselsCross-reactivity of ryanodine receptors with plasma membrane ion channel modulators.Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cellsControl of type I interferon-induced cell death by Orai1-mediated calcium entry in T cellsIncreased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubesNovel inhibitors of the calcineurin/NFATc hub - alternatives to CsA and FK506?Phosphoinositides in Ca(2+) signaling and excitation-contraction coupling in skeletal muscle: an old player and newcomers.Electrical stimulation induces IL-6 in skeletal muscle through extracellular ATP by activating Ca(2+) signals and an IL-6 autocrine loop.Tumor Necrosis Factor Alpha and Insulin-Like Growth Factor 1 Induced Modifications of the Gene Expression Kinetics of Differentiating Skeletal Muscle Cells.Electrical pulse stimulation of cultured skeletal muscle cells as a model for in vitro exercise - possibilities and limitations.ATP released by electrical stimuli elicits calcium transients and gene expression in skeletal muscle.NFAT activation by membrane potential follows a calcium pathway distinct from other activity-related transcription factors in skeletal muscle cells.Sexual Dimorphism in a Reciprocal Interaction of Ryanodine and IP3 Receptors in the Induction of Hyperalgesic Priming.Mitochondrial fusion dynamics is robust in the heart and depends on calcium oscillations and contractile activity.Role of acetylcholine and calcium ions in three vascular contraction models: Angiotensin II, phenylephrine and caffeine.Negative modulation of inositol 1,4,5-trisphosphate type 1 receptor expression prevents dystrophin-deficient muscle cells death.Hypoxia stimulates via separate pathways ERK phosphorylation and NF-kappaB activation in skeletal muscle cells in primary culture.IGF-1 induces IP3 -dependent calcium signal involved in the regulation of myostatin gene expression mediated by NFAT during myoblast differentiation.Plasma cytokine response, lipid peroxidation and NF-kB activation in skeletal muscle following maximum progressive swimming.
P2860
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P2860
NF-kappaB activation by depolarization of skeletal muscle cells depends on ryanodine and IP3 receptor-mediated calcium signals.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
NF-kappaB activation by depola ...... ptor-mediated calcium signals.
@en
type
label
NF-kappaB activation by depola ...... ptor-mediated calcium signals.
@en
prefLabel
NF-kappaB activation by depola ...... ptor-mediated calcium signals.
@en
P2093
P2860
P1476
NF-kappaB activation by depola ...... ptor-mediated calcium signals.
@en
P2093
Enrique Jaimovich
Jorge Hidalgo
José Luis Galaz
Juan Antonio Valdés
M Angélica Carrasco
Mónica Silva
Natalia Puentes
P2860
P304
P356
10.1152/AJPCELL.00320.2006
P577
2007-01-10T00:00:00Z