Epstein-Barr virus BZLF1 gene, a switch from latency to lytic infection, is expressed as an immediate-early gene after primary infection of B lymphocytes.
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Epstein-Barr virus and host cell methylation: regulation of latency, replication and virus reactivationc-FOS suppresses ovarian cancer progression by changing adhesionMeta-analysis of nasopharyngeal carcinoma microarray data explores mechanism of EBV-regulated neoplastic transformationA role for the nucleosome assembly proteins TAF-Iβ and NAP1 in the activation of BZLF1 expression and Epstein-Barr virus reactivationDegradation of phosphorylated p53 by viral protein-ECS E3 ligase complex.AP-1 homolog BZLF1 of Epstein-Barr virus has two essential functions dependent on the epigenetic state of the viral genome.Micro RNAs of Epstein-Barr virus promote cell cycle progression and prevent apoptosis of primary human B cells.CpG-methylation regulates a class of Epstein-Barr virus promoters.Epstein-Barr virus DNase (BGLF5) induces genomic instability in human epithelial cells.Viral response to chemotherapy in endemic burkitt lymphomaNoise cancellation: viral fine tuning of the cellular environment for its own genome replication.The EBV immunoevasins vIL-10 and BNLF2a protect newly infected B cells from immune recognition and eliminationPhase I clinical trial of valacyclovir and standard of care cyclophosphamide in children with endemic Burkitt lymphoma in Malawi.Unexpected patterns of Epstein-Barr virus transcription revealed by a high throughput PCR array for absolute quantification of viral mRNA.Tegument protein control of latent herpesvirus establishment and animation.Susceptibility of human placenta derived mesenchymal stromal/stem cells to human herpesviruses infection.The ZIIR element of the Epstein-Barr virus BZLF1 promoter plays a central role in establishment and maintenance of viral latencyTranscriptome analysis of Kaposi's sarcoma-associated herpesvirus during de novo primary infection of human B and endothelial cells.To be or not IIb: a multi-step process for Epstein-Barr virus latency establishment and consequences for B cell tumorigenesisRNAs in Epstein-Barr virions control early steps of infection.Early T Cell Recognition of B Cells following Epstein-Barr Virus Infection: Identifying Potential Targets for Prophylactic Vaccination.Epstein-Barr viral miRNAs inhibit antiviral CD4+ T cell responses targeting IL-12 and peptide processing.Historical observations contributing insights on etiopathogenesis of rheumatoid arthritis and role of rheumatoid factor.Epstein-Barr virus (EBV) microRNAs: involvement in cancer pathogenesis and immunopathology.Viral manipulation of cellular protein conjugation pathways: The SUMO lesson.Switching of EBV cycles between latent and lytic states.Dynamic Epstein-Barr virus gene expression on the path to B-cell transformation.Modes of infection and oncogenesis by the Epstein-Barr virus.Regulation of Epstein-Barr virus reactivation from latency.Epstein-Barr virus: more than 50 years old and still providing surprises.Visual detection and evaluation of latent and lytic gene expression during Epstein-Barr virus infection using one-step reverse transcription loop-mediated isothermal amplification.Recent advances in understanding Epstein-Barr virus.Shutoff of BZLF1 gene expression is necessary for immortalization of primary B cells by Epstein-Barr virus.Epigenetic histone modification of Epstein-Barr virus BZLF1 promoter during latency and reactivation in Raji cells.EBV lytic infection enhances transformation of B-lymphocytes infected with EBV in the presence of T-lymphocytes.Cellular immediate-early gene expression occurs kinetically upstream of Epstein-Barr virus bzlf1 and brlf1 following cross-linking of the B cell antigen receptor in the Akata Burkitt lymphoma cell line.De novo protein synthesis is required for lytic cycle reactivation of Epstein-Barr virus, but not Kaposi's sarcoma-associated herpesvirus, in response to histone deacetylase inhibitors and protein kinase C agonists.Functions of the Epstein-Barr virus EBNA1 protein in viral reactivation and lytic infection.Epigenetic control of Epstein-Barr virus transcription - relevance to viral life cycle?Epstein-Barr virus in tumor-infiltrating B cells of myasthenia gravis thymoma: an innocent bystander or an autoimmunity mediator?
P2860
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P2860
Epstein-Barr virus BZLF1 gene, a switch from latency to lytic infection, is expressed as an immediate-early gene after primary infection of B lymphocytes.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Epstein-Barr virus BZLF1 gene, ...... ry infection of B lymphocytes.
@en
type
label
Epstein-Barr virus BZLF1 gene, ...... ry infection of B lymphocytes.
@en
prefLabel
Epstein-Barr virus BZLF1 gene, ...... ry infection of B lymphocytes.
@en
P2093
P2860
P356
P1433
P1476
Epstein-Barr virus BZLF1 gene, ...... ry infection of B lymphocytes.
@en
P2093
Dai Iwakiri
Kenzo Takada
Koji Yamamoto
Seiji Maruo
Teru Kanda
Wangrong Wen
P2860
P304
P356
10.1128/JVI.01416-06
P407
P577
2006-11-01T00:00:00Z