ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s.

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Group 2 Innate Lymphoid Cells in Pulmonary Immunity and Tissue Homeostasis.

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Q53818897-D1B80148-1255-4102-B644-347888C0B504

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ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s.

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ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s.

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ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s.

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ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s.

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P1476

ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s.

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P2093

A I Sperling

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C L Hrusch

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D Bryazka

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I Noth

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J Casaos

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K A M Mills

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K M Blaine

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M E Strek

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M R Jaffery

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M Y Tjota

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P2860

Idiopathic pulmonary fibrosis: from epithelial injury to biomarkers--insights from the bench side

ICOS co-stimulatory receptor is essential for T-cell activation and function

Unifying concepts in CD28, ICOS and CTLA4 co-receptor signalling

An official ATS/ERS/JRS/ALAT statement: idiopathic pulmonary fibrosis: evidence-based guidelines for diagnosis and management

Mechanisms of fibrosis: therapeutic translation for fibrotic disease

Group 2 innate lymphoid cell production of IL-5 is regulated by NKT cells during influenza virus infection

ICOS/ICOSL interaction is required for CD4+ invariant NKT cell function and homeostatic survival.

Pulmonary permeability assessed by fluorescent-labeled dextran instilled intranasally into mice with LPS-induced acute lung injury.

The Berlin definition of ARDS: an expanded rationale, justification, and supplementary material.

New therapeutic targets in idiopathic pulmonary fibrosis. Aiming to rein in runaway wound-healing responses.

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mi.2017.42

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61-70

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scholarly article

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10.1038/MI.2017.42

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Ayodeji Adegunsoye

Jesse Williams

Catherine Bonham

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2017-05-10T00:00:00Z

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28488693

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5681437

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ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s.

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ICOS protects against mortality from acute lung injury through activation of IL-5+ ILC2s.

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