Angiotensin II-induced NADPH oxidase activation impairs insulin signaling in skeletal muscle cells. - Wikidata - wikimedia - TriplyDB

Angiotensin II-induced NADPH oxidase activation impairs insulin signaling in skeletal muscle cells.

Angiotensin II-induced NADPH oxidase activation impairs insulin signaling in skeletal muscle cells.

http://www.wikidata.org/entity/Q40230599

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Non-alcoholic fatty liver disease: what has changed in the treatment since the beginning?Mitochondrial dysfunction in heart failure Functional sympatholysis in hypertension Modulation of glucose metabolism by the renin-angiotensin-aldosterone system Molecular Mechanisms of Obesity-Induced Osteoporosis and Muscle Atrophy NADPH oxidase activation is required in reactive oxygen species generation and cell transformation induced by hexavalent chromium Elevated hydrogen peroxide and decreased catalase and glutathione peroxidase protection are associated with aging sarcopenia Oxygen consumption and usage during physical exercise: the balance between oxidative stress and ROS-dependent adaptive signaling Angiotensin II-induced skeletal muscle insulin resistance mediated by NF-kappaB activation via NADPH oxidase Bridging Type 2 Diabetes and Alzheimer's Disease: Assembling the Puzzle Pieces in the Quest for the Molecules With Therapeutic and Preventive Potential Reactive Oxygen Species/Nitric Oxide Mediated Inter-Organ Communication in Skeletal Muscle Wasting Diseases.Angiotensin-(1-7) recruits muscle microvasculature and enhances insulin's metabolic action via mas receptor Angiotensin II inhibits insulin-stimulated GLUT4 translocation and Akt activation through tyrosine nitration-dependent mechanisms.Oxidative Stress in Human Aorta of Patients with Advanced Aortoiliac Occlusive Disease.Meal-related increases in microvascular vasomotion are impaired in obese individuals: a potential mechanism in the pathogenesis of obesity-related insulin resistance.Angiotensin receptor blocker/diuretic combination preserves insulin responses in obese hypertensives.The effects of palmitate on hepatic insulin resistance are mediated by NADPH Oxidase 3-derived reactive oxygen species through JNK and p38MAPK pathways Angiotensin-induced EGF receptor transactivation inhibits insulin signaling in C9 hepatic cells.Angiotensin II modulates mouse skeletal muscle resting conductance to chloride and potassium ions and calcium homeostasis via the AT1 receptor and NADPH oxidase Oxidative stress and the etiology of insulin resistance and type 2 diabetes.The Impact of Overnutrition on Insulin Metabolic Signaling in the Heart and the Kidney Angiotensin II induced catabolic effect and muscle atrophy are redox dependent.Metabolic syndrome: definitions and controversies.Middle age aggravates myocardial ischemia through surprising upholding of complex II activity, oxidative stress, and reduced coronary perfusion.Skeletal muscle insulin resistance in salt-sensitive hypertension: role of angiotensin II activation of NFκB.Sirtuin3 Dysfunction Is the Key Determinant of Skeletal Muscle Insulin Resistance by Angiotensin II Unbreak my heart: targeting mitochondrial autophagy in diabetic cardiomyopathy Direct inhibition by angiotensin II of insulin-dependent glucose transport activity in mammalian skeletal muscle involves a ROS-dependent mechanism.Activation of mTOR/p70S6 kinase by ANG II inhibits insulin-stimulated endothelial nitric oxide synthase and vasodilation.Chronic renin inhibition with aliskiren improves glucose tolerance, insulin sensitivity, and skeletal muscle glucose transport activity in obese Zucker rats The metabolic syndrome, oxidative stress, environment, and cardiovascular disease: the great exploration.Mechanisms of Cachexia in Chronic Disease States Exploring metabolic dysfunction in chronic kidney disease.The renin angiotensin aldosterone system and insulin resistance in humans.Differential predictors of insulin resistance in nondiabetic salt-resistant and salt-sensitive subjects.Angiotensin 1-7 as means to prevent the metabolic syndrome: lessons from the fructose-fed rat model.AT1 receptor blocker losartan protects against mechanical ventilation-induced diaphragmatic dysfunction Systemic oxidative stress is associated with lower aerobic capacity and impaired skeletal muscle energy metabolism in patients with metabolic syndrome.Low-dose spironolactone reduces reactive oxygen species generation and improves insulin-stimulated glucose transport in skeletal muscle in the TG(mRen2)27 rat.Improvement of insulin sensitivity by antagonism of the renin-angiotensin system.

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P2860

Angiotensin II-induced NADPH oxidase activation impairs insulin signaling in skeletal muscle cells.

http://www.wikidata.org/entity/Q40230599

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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Angiotensin II-induced NADPH o ...... ling in skeletal muscle cells.

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Angiotensin II-induced NADPH o ...... ling in skeletal muscle cells.

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Angiotensin II-induced NADPH o ...... ling in skeletal muscle cells.

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Angiotensin II-induced NADPH o ...... aling in skeletal muscle cells

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P2093

Camila Manrique

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Craig S Stump

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Grace M-E Uptergrove

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Heping Gong

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James R Sowers

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Nicholas Szary

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Ravi Nistala

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Suzanne E Clark

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Yongzhong Wei

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P2860

A region C-terminal to the proline-rich core of p47phox regulates activation of the phagocyte NADPH oxidase by interacting with the C-terminal SH3 domain of p67phox

Protein kinase B kinases that mediate phosphatidylinositol 3,4,5-trisphosphate-dependent activation of protein kinase B

p22phox is a critical component of the superoxide-generating NADH/NADPH oxidase system and regulates angiotensin II-induced hypertrophy in vascular smooth muscle cells

Dual role of phosphatidylinositol-3,4,5-trisphosphate in the activation of protein kinase B

Prevalence of the metabolic syndrome among US adults: findings from the third National Health and Nutrition Examination Survey

NAD(P)H oxidase: role in cardiovascular biology and disease

Effects of an angiotensin-converting-enzyme inhibitor, ramipril, on cardiovascular events in high-risk patients

Molecular dynamics study on the ligand recognition by tandem SH3 domains of p47phox, regulating NADPH oxidase activity.

Role of gp91phox (Nox2)-containing NAD(P)H oxidase in angiogenesis in response to hindlimb ischemia.

Role of the adipocyte, free fatty acids, and ectopic fat in pathogenesis of type 2 diabetes mellitus: peroxisomal proliferator-activated receptor agonists provide a rational therapeutic approach.

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35137-35146

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10.1074/JBC.M601320200

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46

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281

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E Matthew Morris

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2006-09-17T00:00:00Z

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16982630

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