BCR-ABL fusion regions as a source of multiple leukemia-specific CD8+ T-cell epitopes. - Wikidata - wikimedia - TriplyDB

BCR-ABL fusion regions as a source of multiple leukemia-specific CD8+ T-cell epitopes.

BCR-ABL fusion regions as a source of multiple leukemia-specific CD8+ T-cell epitopes.

http://www.wikidata.org/entity/Q40239641

about

T cell receptor mimic antibodies for cancer therapy Immunotherapy in acute leukemia Vaccines against human carcinomas: strategies to improve antitumor immune responses.The role of peptide and DNA vaccines in myeloid leukemia immunotherapy Translational mini-review series on vaccines: Peptide vaccines for myeloid leukaemias.Identification of T-cell epitopes for cancer immunotherapy.Leukemia-associated antigens as target structures for a specific immunotherapy in chronic myeloid leukemia.Immunotherapy for myeloid leukemias: current status and future directions.The biological basis for immunotherapy in patients with chronic myelogenous leukemia.Introduction of neutralizing immunogenicity index to the rational design of MERS coronavirus subunit vaccines.Immunology and immunotherapy of chronic myeloid leukemia.Sequence and expression of the chicken membrane-associated phospholipases A1 alpha (LIPH) and beta (LIPI).Human leukocyte antigen class I alleles and the risk of chronic myelogenous leukemia: a meta-analysis.Change in expression pattern of TCR-CD3 complex in patients with multiple myeloma.

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P2860

BCR-ABL fusion regions as a source of multiple leukemia-specific CD8+ T-cell epitopes.

http://www.wikidata.org/entity/Q40239641

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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name

BCR-ABL fusion regions as a source of multiple leukemia-specific CD8+ T-cell epitopes.

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type

label

BCR-ABL fusion regions as a source of multiple leukemia-specific CD8+ T-cell epitopes.

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prefLabel

BCR-ABL fusion regions as a source of multiple leukemia-specific CD8+ T-cell epitopes.

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BCR-ABL fusion regions as a source of multiple leukemia-specific CD8+ T-cell epitopes.

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Bres-Vloemans SA

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Camps M

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Drijfhout JW

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Huijbers IJ

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Kessler JH

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Leeksma OC

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Melief CJ

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Mulder A

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Offringa R

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Ossendorp F

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P2860

Subunit arrangement in the human 20S proteasome

Association of HLA Class I and Class II genes with bcr-abl transcripts in leukemia patients with t(9;22) (q34;q11)

Direct evidence that leukemic cells present HLA-associated immunogenic peptides derived from the BCR-ABL b3a2 fusion protein

Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells

Multiple BCR-ABL kinase domain mutations confer polyclonal resistance to the tyrosine kinase inhibitor imatinib (STI571) in chronic phase and blast crisis chronic myeloid leukemia

SYFPEITHI: database for MHC ligands and peptide motifs

Clinical resistance to STI-571 cancer therapy caused by BCR-ABL gene mutation or amplification

Discrete cleavage motifs of constitutive and immunoproteasomes revealed by quantitative analysis of cleavage products.

The law of mass action governs antigen-stimulated cytolytic activity of CD8+ cytotoxic T lymphocytes.

Immunodominance in major histocompatibility complex class I-restricted T lymphocyte responses.

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scholarly article

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10.1038/SJ.LEU.2404354

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10

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20

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Peter van Veelen

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2006-08-24T00:00:00Z

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6856731

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