Epigenetic silencing of the c-fms locus during B-lymphopoiesis occurs in discrete steps and is reversible.
about
Rapid, solid-phase based automated analysis of chromatin structure and transcription factor occupancy in living eukaryotic cellsPrecancerous stem cells have the potential for both benign and malignant differentiationCSF-1 receptor signaling in myeloid cellsThe function of the conserved regulatory element within the second intron of the mammalian Csf1r locus.A distant upstream locus control region is critical for expression of the Kit receptor gene in mast cellsEpigenetic characterization of hematopoietic stem cell differentiation using miniChIP and bisulfite sequencing analysis.Lineage specification and plasticity in CD19- early B cell precursors.B cells and osteoblast and osteoclast development.Clonally related follicular lymphomas and histiocytic/dendritic cell sarcomas: evidence for transdifferentiation of the follicular lymphoma clone.PU.1 expression is modulated by the balance of functional sense and antisense RNAs regulated by a shared cis-regulatory element.Action at a distance: epigenetic silencing of large chromosomal regions in carcinogenesis.Transcriptional repression of Gata3 is essential for early B cell commitment.Hereditary diffuse gastric cancer and lost cell polarity: a short path to cancer.Early chromatin unfolding by RUNX1: a molecular explanation for differential requirements during specification versus maintenance of the hematopoietic gene expression program.Epigenetic mechanisms regulating normal and malignant haematopoiesis: new therapeutic targets for clinical medicine.Epigenetic regulation of B lymphocyte differentiation, transdifferentiation, and reprogramming.Chromatin programming by developmentally regulated transcription factors: lessons from the study of haematopoietic stem cell specification and differentiation.Protein-coding cis-natural antisense transcripts have high and broad expression in Arabidopsis.Interleukin-7-induced Stat-5 acts in synergy with Flt-3 signaling to stimulate expansion of hematopoietic progenitor cells.Formation of an active tissue-specific chromatin domain initiated by epigenetic marking at the embryonic stem cell stage.A two-step, PU.1-dependent mechanism for developmentally regulated chromatin remodeling and transcription of the c-fms gene.The mechanism of repression of the myeloid-specific c-fms gene by Pax5 during B lineage restrictionThe Pu.1 locus is differentially regulated at the level of chromatin structure and noncoding transcription by alternate mechanisms at distinct developmental stages of hematopoiesis.Protein kinase A inhibition of macrophage maturation is accompanied by an increase in DNA methylation of the colony-stimulating factor 1 receptor gene.Aberrant expression of CD19 in AML with t(8;21) involves a poised chromatin structure and PAX5.
P2860
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P2860
Epigenetic silencing of the c-fms locus during B-lymphopoiesis occurs in discrete steps and is reversible.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Epigenetic silencing of the c- ...... crete steps and is reversible.
@en
type
label
Epigenetic silencing of the c- ...... crete steps and is reversible.
@en
prefLabel
Epigenetic silencing of the c- ...... crete steps and is reversible.
@en
P2860
P50
P356
P1433
P1476
Epigenetic silencing of the c- ...... screte steps and is reversible
@en
P2093
Alexandra Schebesta
Pascal Lefevre
P2860
P304
P356
10.1038/SJ.EMBOJ.7600421
P407
P577
2004-10-14T00:00:00Z