Receptor (SLAM [CD150]) recognition and the V protein sustain swift lymphocyte-based invasion of mucosal tissue and lymphatic organs by a morbillivirus.
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Early target cells of measles virus after aerosol infection of non-human primatesPredominant infection of CD150+ lymphocytes and dendritic cells during measles virus infection of macaquesMorbillivirus Experimental Animal Models: Measles Virus Pathogenesis Insights from Canine Distemper VirusThe nonstructural proteins of Nipah virus play a key role in pathogenicity in experimentally infected animals.Canine distemper virus persistence in demyelinating encephalitis by swift intracellular cell-to-cell spread in astrocytes is controlled by the viral attachment protein.Human parainfluenza virus type 2 V protein inhibits interferon production and signaling and is required for replication in non-human primates.Measles virus infection of alveolar macrophages and dendritic cells precedes spread to lymphatic organs in transgenic mice expressing human signaling lymphocytic activation molecule (SLAM, CD150)Measles virus selectively blind to signaling lymphocytic activation molecule (SLAM; CD150) is attenuated and induces strong adaptive immune responses in rhesus monkeys.Comparative pathogenesis of Alkhumra hemorrhagic fever and Kyasanur forest disease viruses in a mouse model.Cross-species transmission of canine distemper virus-an updateGenome Sequences of Three Vaccine Strains and Two Wild-Type Canine Distemper Virus Strains from a Recent Disease Outbreak in South Africa.Elements in the canine distemper virus M 3' UTR contribute to control of replication efficiency and virulenceDistinct and overlapping roles of Nipah virus P gene products in modulating the human endothelial cell antiviral response.Early events following experimental infection with Peste-Des-Petits ruminants virus suggest immune cell targetingMorbillivirus v proteins exhibit multiple mechanisms to block type 1 and type 2 interferon signalling pathwaysThe V protein of canine distemper virus is required for virus replication in human epithelial cells.The immunomodulating V and W proteins of Nipah virus determine disease course.Canine distemper virus in the Serengeti ecosystem: molecular adaptation to different carnivore species.Delineating morbillivirus entry, dissemination and airborne transmission by studying in vivo competition of multicolor canine distemper viruses in ferrets.Attenuation of V- or C-defective measles viruses: infection control by the inflammatory and interferon responses of rhesus monkeys.Measles virus blind to its epithelial cell receptor remains virulent in rhesus monkeys but cannot cross the airway epithelium and is not shed.Region between the canine distemper virus M and F genes modulates virulence by controlling fusion protein expression.Virus growth and antibody responses following respiratory tract infection of ferrets and mice with WT and P/V mutants of the paramyxovirus Simian Virus 5.Studies on the paramyxovirus accessory genes by reverse genetics in the Sendai virus-mouse system.Nipah Virus C and W Proteins Contribute to Respiratory Disease in Ferrets.Paramyxovirus disruption of interferon signal transduction: STATus reportMorbillivirus control of the interferon response: relevance of STAT2 and mda5 but not STAT1 for canine distemper virus virulence in ferrets.Reverse genetics of Mononegavirales: How they work, new vaccines, and new cancer therapeuticsMeasles vaccination of nonhuman primates provides partial protection against infection with canine distemper virus.Modulation of Host Immunity by the Human Metapneumovirus.Measles virus breaks through epithelial cell barriers to achieve transmission.Emergence of canine distemper virus strains with modified molecular signature and enhanced neuronal tropism leading to high mortality in wild carnivores.Canine distemper virus epithelial cell infection is required for clinical disease but not for immunosuppression.Canine distemper virus matrix protein influences particle infectivity, particle composition, and envelope distribution in polarized epithelial cells and modulates virulence.Canine distemper virus selectively inhibits apoptosis progression in infected immune cells.In Vitro Exposure of Harbor Seal Immune Cells to Aroclor 1260 Alters Phocine Distemper Virus Replication.Early cytokine mRNA expression profiles predict Morbillivirus disease outcome in ferrets.Immunopathogenic and neurological mechanisms of canine distemper virus.Measles virus infection of SLAM (CD150) knockin mice reproduces tropism and immunosuppression in human infectionDisease duration determines canine distemper virus neurovirulence.
P2860
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P2860
Receptor (SLAM [CD150]) recognition and the V protein sustain swift lymphocyte-based invasion of mucosal tissue and lymphatic organs by a morbillivirus.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Receptor (SLAM [CD150]) recogn ...... tic organs by a morbillivirus.
@en
type
label
Receptor (SLAM [CD150]) recogn ...... tic organs by a morbillivirus.
@en
prefLabel
Receptor (SLAM [CD150]) recogn ...... tic organs by a morbillivirus.
@en
P2860
P356
P1433
P1476
Receptor (SLAM [CD150]) recogn ...... tic organs by a morbillivirus.
@en
P2093
Veronika von Messling
P2860
P304
P356
10.1128/JVI.00357-06
P407
P577
2006-06-01T00:00:00Z