Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression.
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Oxidative Stress Signaling in Alzheimers DiseaseRAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunctionMitochondria, calcium and cell death: a deadly triad in neurodegenerationThe Rationale for Insulin Therapy in Alzheimer's DiseaseIntracellular Calcium Dysregulation: Implications for Alzheimer's DiseaseMitochondrial Dysfunction in Alzheimer's Disease and the Rationale for Bioenergetics Based TherapiesCross Talk of Proteostasis and Mitostasis in Cellular Homeodynamics, Ageing, and DiseaseA Potential Alternative against Neurodegenerative Diseases: PhytodrugsKilling Me Softly: Connotations to Unfolded Protein Response and Oxidative Stress in Alzheimer's DiseaseAlterations in Mitochondrial Quality Control in Alzheimer's DiseaseAstaxanthin as a Potential Neuroprotective Agent for Neurological DiseasesMitochondrial Dysfunction Contributes to the Pathogenesis of Alzheimer's DiseaseDiverse molecular targets for therapeutic strategies in Alzheimer's diseaseShift in brain metabolism in late onset Alzheimer's disease: implications for biomarkers and therapeutic interventionsOxidative stress and cerebral endothelial cells: regulation of the blood-brain-barrier and antioxidant based interventionsMitochondria-Targeted Protective Compounds in Parkinson's and Alzheimer's DiseasesReevaluating Metabolism in Alzheimer's Disease from the Perspective of the Astrocyte-Neuron Lactate Shuttle ModelMetabolic disturbances in diseases with neurological involvementAmyloid β precursor protein as a molecular target for amyloid β--induced neuronal degeneration in Alzheimer's diseaseOxidized cholesterol as the driving force behind the development of Alzheimer's diseaseMitochondrial permeability transition pore is a potential drug target for neurodegenerationMitochondrial glutathione: features, regulation and role in diseaseAltered brain energetics induces mitochondrial fission arrest in Alzheimer's Disease.Defects in mitochondrial dynamics and metabolomic signatures of evolving energetic stress in mouse models of familial Alzheimer's diseaseMolecular Basis of Substrate Recognition and Degradation by Human Presequence ProteaseDynamics of Human Mitochondrial Complex I Assembly: Implications for Neurodegenerative DiseasesVitamin A and Retinoids as Mitochondrial ToxicantsAbnormal interaction between the mitochondrial fission protein Drp1 and hyperphosphorylated tau in Alzheimer's disease neurons: implications for mitochondrial dysfunction and neuronal damageMitochondrial defects and oxidative stress in Alzheimer disease and Parkinson diseaseMitochondrial fragmentation in neurodegenerationDynamin-related protein 1 and mitochondrial fragmentation in neurodegenerative diseasesParkin-mediated mitophagy in mutant hAPP neurons and Alzheimer's disease patient brainsMorphological and bioenergetic demands underlying the mitophagy in post-mitotic neurons: the pink-parkin pathwayExamining the interactome of huperzine A by magnetic biopanningGeniposide Protects Primary Cortical Neurons against Oligomeric Aβ1-42-Induced Neurotoxicity through a Mitochondrial PathwayMitochondria-specific accumulation of amyloid β induces mitochondrial dysfunction leading to apoptotic cell deathMitochondrial dynamics--fusion, fission, movement, and mitophagy--in neurodegenerative diseasesPathogenesis of Alzheimer's diseaseMitochondria are related to synaptic pathology in Alzheimer's disease.Amyloid-beta and mitochondria in aging and Alzheimer's disease: implications for synaptic damage and cognitive decline
P2860
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P2860
Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Mitochondria are a direct site ...... damage in disease progression.
@en
type
label
Mitochondria are a direct site ...... damage in disease progression.
@en
prefLabel
Mitochondria are a direct site ...... damage in disease progression.
@en
P2093
P356
P1476
Mitochondria are a direct site ...... damage in disease progression.
@en
P2093
Byung S Park
Edward Henson
Maria Manczak
P Hemachandra Reddy
Thimmappa S Anekonda
P304
P356
10.1093/HMG/DDL066
P50
P577
2006-03-21T00:00:00Z