Control of human papillomavirus type 11 origin of replication by the E2 family of transcription regulatory proteins.
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Brd4 links chromatin targeting to HPV transcriptional silencingA Structural Basis for BRD2/4-Mediated Host Chromatin Interaction and Oligomer Assembly of Kaposi Sarcoma-Associated Herpesvirus and Murine Gammaherpesvirus LANA ProteinsVariant upstream regulatory region sequences differentially regulate human papillomavirus type 16 DNA replication throughout the viral life cycle.Competition for DNA binding sites between the short and long forms of E2 dimers underlies repression in bovine papillomavirus type 1 DNA replication control.Mechanisms of human papillomavirus E2-mediated repression of viral oncogene expression and cervical cancer cell growth inhibition.Alleviation of human papillomavirus E2-mediated transcriptional repression via formation of a TATA binding protein (or TFIID)-TFIIB-RNA polymerase II-TFIIF preinitiation complex.Human TATA binding protein inhibits human papillomavirus type 11 DNA replication by antagonizing E1-E2 protein complex formation on the viral origin of replication.Control of HPV 18 DNA replication by cellular and viral transcription factors.Human papillomavirus type 31 replication modes during the early phases of the viral life cycle depend on transcriptional and posttranscriptional regulation of E1 and E2 expression.Replication and partitioning of papillomavirus genomes.Human papillomavirus type 31 oncoproteins E6 and E7 are required for the maintenance of episomes during the viral life cycle in normal human keratinocytes.Replicative intermediates of human papillomavirus type 11 in laryngeal papillomas: site of replication initiation and direction of replication.Identification and analysis of papillomavirus E2 protein binding sites in the human genome.E1 protein of human papillomavirus type 1a is sufficient for initiation of viral DNA replication.Genetic analysis of the activation domain of bovine papillomavirus protein E2: its role in transcription and replication.A papillomavirus E2 phosphorylation mutant exhibits normal transient replication and transcription but is defective in transformation and plasmid retentionProductive Lifecycle of Human Papillomaviruses that Depends Upon Squamous Epithelial Differentiation.Mechanisms of human papillomavirus-induced oncogenesis.Human papillomavirus type 16 E5 protein as a therapeutic target.Comprehensive comparison of the interaction of the E2 master regulator with its cognate target DNA sites in 73 human papillomavirus types by sequence statistics.Variation of human papillomavirus type 6 (HPV-6) and HPV-11 genomes sampled throughout the world.Human papillomavirus type 11 E2 proteins repress the homologous E6 promoter by interfering with the binding of host transcription factors to adjacent elements.The human papillomavirus type 16 E2 transcription factor binds with low cooperativity to two flanking sites and represses the E6 promoter through displacement of Sp1 and TFIID.Human papillomavirus infections: warts or cancer?Characterization of the human papillomavirus E2 protein: evidence of trans-activation and trans-repression in cervical keratinocytes.Two E2 binding sites alone are sufficient to function as the minimal origin of replication of human papillomavirus type 18 DNA.Orientation of a novel DNA binding site affects human papillomavirus-mediated transcription and replication.Transcription factor YY1 represses cell-free replication from human papillomavirus origins.Identification of the E9/E2C cDNA and functional characterization of the gene product reveal a new repressor of transcription and replication in cottontail rabbit papillomavirus.Tumor suppressor or oncogene? A critical role of the human papillomavirus (HPV) E2 protein in cervical cancer progression.Mapping of betapapillomavirus human papillomavirus 5 transcription and characterization of viral-genome replication function.Human papillomavirus DNA replication compartments in a transient DNA replication system.The carboxyl-terminal region of the human papillomavirus type 16 E1 protein determines E2 protein specificity during DNA replication.The E8E2C protein, a negative regulator of viral transcription and replication, is required for extrachromosomal maintenance of human papillomavirus type 31 in keratinocytes.The hinge of the human papillomavirus type 11 E2 protein contains major determinants for nuclear localization and nuclear matrix associationThe E8 domain confers a novel long-distance transcriptional repression activity on the E8E2C protein of high-risk human papillomavirus type 31.Development of a cellular assay system to study the genome replication of high- and low-risk mucosal and cutaneous human papillomavirusesDifferential requirements for conserved E2 binding sites in the life cycle of oncogenic human papillomavirus type 31Chaperone proteins abrogate inhibition of the human papillomavirus (HPV) E1 replicative helicase by the HPV E2 protein.cis-Acting components of human papillomavirus (HPV) DNA replication: linker substitution analysis of the HPV type 11 origin.
P2860
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P2860
Control of human papillomavirus type 11 origin of replication by the E2 family of transcription regulatory proteins.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Control of human papillomaviru ...... scription regulatory proteins.
@en
type
label
Control of human papillomaviru ...... scription regulatory proteins.
@en
prefLabel
Control of human papillomaviru ...... scription regulatory proteins.
@en
P2093
P2860
P1433
P1476
Control of human papillomaviru ...... nscription regulatory proteins
@en
P2093
P2860
P304
P407
P577
1992-09-01T00:00:00Z