Inorganic arsenic compounds cause oxidative damage to DNA and protein by inducing ROS and RNS generation in human keratinocytes.
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Candidate single nucleotide polymorphism markers for arsenic responsiveness of protein targetsLow concentration of arsenite exacerbates UVR-induced DNA strand breaks by inhibiting PARP-1 activityInhibition of poly(ADP-ribose) polymerase-1 by arsenite interferes with repair of oxidative DNA damageArsenic-induced malignant transformation of human keratinocytes: involvement of Nrf2Arsenite interacts selectively with zinc finger proteins containing C3H1 or C4 motifsEnhanced ROS production and redox signaling with combined arsenite and UVA exposure: contribution of NADPH oxidaseProduction of superoxide anions by keratinocytes initiates P. acnes-induced inflammation of the skin.Oncogenomic disruptions in arsenic-induced carcinogenesis.Role and mechanism of arsenic in regulating angiogenesis.Aberrantly Expressed Genes in HaCaT Keratinocytes Chronically Exposed to Arsenic Trioxide.Emodin Induces Apoptotic Death in Murine Myelomonocytic Leukemia WEHI-3 Cells In Vitro and Enhances Phagocytosis in Leukemia Mice In VivoThe NRF2-KEAP1 pathway is an early responsive gene network in arsenic exposed lymphoblastoid cells.Nicotinamide enhances repair of arsenic and ultraviolet radiation-induced DNA damage in HaCaT keratinocytes and ex vivo human skinA Curcumin Derivative That Inhibits Vinyl Carbamate-Induced Lung Carcinogenesis via Activation of the Nrf2 Protective Response.GSTM1 and APE1 genotypes affect arsenic-induced oxidative stress: a repeated measures studyVDAC1: from structure to cancer therapy.Dual actions involved in arsenite-induced oxidative DNA damage.As(III) inhibits ultraviolet radiation-induced cyclobutane pyrimidine dimer repair via generation of nitric oxide in human keratinocytes.Genetic susceptibility to arsenic-induced skin lesions and health effects: a reviewArsenite-induced ROS/RNS generation causes zinc loss and inhibits the activity of poly(ADP-ribose) polymerase-1.Melanocytes and keratinocytes have distinct and shared responses to ultraviolet radiation and arsenic.Melatonin protects against arsenic trioxide-induced liver injury by the upregulation of Nrf2 expression through the activation of PI3K/AKT pathway.Natural Antioxidants Against Arsenic-Induced Genotoxicity.Arsenic trioxide induces ROS activity and DNA damage, leading to G0/G1 extension in skin fibroblasts through the ATM-ATR-associated Chk pathway.Interdependent genotoxic mechanisms of monomethylarsonous acid: role of ROS-induced DNA damage and poly(ADP-ribose) polymerase-1 inhibition in the malignant transformation of urothelial cellsBRCA2-dependent homologous recombination is required for repair of Arsenite-induced replication lesions in mammalian cells.Susceptibility to arsenic-induced skin lesions from polymorphisms in base excision repair genes.Arsenic levels in cutaneous appendicular organs are correlated with digitally evaluated hyperpigmented skin of the forehead but not the sole in Bangladesh residents.Electrozymographic evaluation of the attenuation of arsenic induced degradation of hepatic SOD, catalase in an in vitro assay system by pectic polysaccharides of Momordica charantia in combination with curcumin.Normobaric hyperoxia delays and attenuates early nitric oxide production in focal cerebral ischemic rats.Evaluation of gene expression changes in human primary uroepithelial cells following 24-hr exposures to inorganic arsenic and its methylated metabolites.A critical role for IkappaB kinase beta in metallothionein-1 expression and protection against arsenic toxicity.VDAC1 cysteine residues: topology and function in channel activity and apoptosis.Global gene expression profiling of hyperkeratotic skin lesions from inner Mongolians chronically exposed to arsenic.Arsenite induces DNA damage via mitochondrial ROS and induction of mitochondrial permeability transition.Maternal/fetal metabolomes appear to mediate the impact of arsenic exposure on birth weight: A pilot study.Deciphering the molecular events during arsenic induced transcription signal cascade activation in cellular milieu.Ellagic acid mitigates arsenic-trioxide-induced mitochondrial dysfunction and cytotoxicity in SH-SY5Y cells.Protection of Taurine Against Arsenic-Induced DNA Damage of Mice Kidneys.Individual susceptibility to arsenic-induced diseases: the role of host genetics, nutritional status, and the gut microbiome.
P2860
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P2860
Inorganic arsenic compounds cause oxidative damage to DNA and protein by inducing ROS and RNS generation in human keratinocytes.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Inorganic arsenic compounds ca ...... ration in human keratinocytes.
@en
type
label
Inorganic arsenic compounds ca ...... ration in human keratinocytes.
@en
prefLabel
Inorganic arsenic compounds ca ...... ration in human keratinocytes.
@en
P2093
P1476
Inorganic arsenic compounds ca ...... ration in human keratinocytes.
@en
P2093
Ke Jian Liu
Laurie G Hudson
P2860
P2888
P304
P356
10.1007/S11010-005-8227-Y
P577
2005-11-01T00:00:00Z