Nitric oxide induces [Ca2+]i oscillations in pituitary GH3 cells: involvement of IDR and ERG K+ currents. - Wikidata - wikimedia - TriplyDB

Nitric oxide induces [Ca2+]i oscillations in pituitary GH3 cells: involvement of IDR and ERG K+ currents.

Nitric oxide induces [Ca2+]i oscillations in pituitary GH3 cells: involvement of IDR and ERG K+ currents.

http://www.wikidata.org/entity/Q40365067

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Nitric oxide mediates stretch-induced Ca2+ release via activation of phosphatidylinositol 3-kinase-Akt pathway in smooth muscle Nitric oxide has differential effects on currents in different subsets of Manduca sexta antennal lobe neurons.Acetylcholine promotes Ca2+ and NO-oscillations in adipocytes implicating Ca2+→NO→cGMP→cADP-ribose→Ca2+ positive feedback loop--modulatory effects of norepinephrine and atrial natriuretic peptide MicroRNA-103-1 selectively downregulates brain NCX1 and its inhibition by anti-miRNA ameliorates stroke damage and neurological deficits.Concepts of neural nitric oxide-mediated transmission.Skeletal muscle insulin resistance: role of inflammatory cytokines and reactive oxygen species.Lipid and insulin infusion-induced skeletal muscle insulin resistance is likely due to metabolic feedback and not changes in IRS-1, Akt, or AS160 phosphorylation Intracellular and plasma membrane-initiated pathways involved in the [Ca2+]i elevations induced by iodothyronines (T3 and T2) in pituitary GH3 cells.From clinical insights to new therapies.

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P2860

Nitric oxide induces [Ca2+]i oscillations in pituitary GH3 cells: involvement of IDR and ERG K+ currents.

http://www.wikidata.org/entity/Q40365067

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年论文

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2005年论文

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name

Nitric oxide induces [Ca2+]i o ...... nt of IDR and ERG K+ currents.

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Nitric oxide induces [Ca2+]i o ...... nt of IDR and ERG K+ currents.

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Nitric oxide induces [Ca2+]i o ...... nt of IDR and ERG K+ currents.

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American Journal of Physiology - Cell Physiology

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Nitric oxide induces [Ca2+]i o ...... nt of IDR and ERG K+ currents.

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Agnese Secondo

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Anna Pannaccione

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Gianfranco Di Renzo

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Lucio Annunziato

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Luigi Formisano

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Mauro Cataldi

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Rossana Sirabella

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P2860

Modulation of Kv3 potassium channels expressed in CHO cells by a nitric oxide-activated phosphatase

Phoneutria nigriventer toxin Tx3-1 blocks A-type K+ currents controlling Ca2+ oscillation frequency in GH3 cells.

Modulation of ion channels by reactive oxygen and nitrogen species: a pathophysiological role in brain aging?

Nitric oxide. A macrophage product responsible for cytostasis and respiratory inhibition in tumor target cells.

Modulation of potassium channel function by methionine oxidation and reduction

Calcium spiking.

Calcium channels, stores, and oscillations.

Electrophysiology of excitable endocrine cells.

Involvement of PI3'-K, mitogen-activated protein kinase and protein kinase B in the up-regulation of the expression of nNOSalpha and nNOSbeta splicing variants induced by PRL-receptor activation in GH3 cells.

Spontaneous calcium oscillations control c-fos transcription via the serum response element in neuroendocrine cells.

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C233-43

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10.1152/AJPCELL.00231.2005

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7557338

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16207796

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