FLIP is frequently expressed in endometrial carcinoma and has a role in resistance to TRAIL-induced apoptosis.
about
Knockdown of MTDH sensitizes endometrial cancer cells to cell death induction by death receptor ligand TRAIL and HDAC inhibitor LBH589 co-treatment.HBx-induced NF-κB signaling in liver cells is potentially mediated by the ternary complex of HBx with p22-FLIP and NEMO.B lymphocytes are resistant to death receptor 5-induced apoptosis.KSR1 is overexpressed in endometrial carcinoma and regulates proliferation and TRAIL-induced apoptosis by modulating FLIP levels.A cell-based high-throughput screen to identify synergistic TRAIL sensitizers.Cellular FLICE-like inhibitory protein (C-FLIP): a novel target for cancer therapy.TNF-related apoptosis-inducing ligand as a therapeutic agent in autoimmunity and cancer.The synergistic apoptotic interaction of Indole-3-Carbinol and Genistein with TRAIL on endometrial cancer cells.CK2beta is expressed in endometrial carcinoma and has a role in apoptosis resistance and cell proliferation.Endometrial carcinoma: molecular alterations involved in tumor development and progression.The three Rs along the TRAIL: resistance, re-sensitization and reactive oxygen species (ROS).Molecular pathology of endometrial carcinoma.Osteoblasts display different responsiveness to TRAIL-induced apoptosis during their differentiation process.Combination of Vorinostat and caspase-8 inhibition exhibits high anti-tumoral activity on endometrial cancer cells.Sorafenib induces endometrial carcinoma apoptosis by inhibiting Elk-1-dependent Mcl-1 transcription and inducing Akt/GSK3β-dependent protein degradation.Immunohistochemical features of post-radiation vaginal recurrences of endometrioid carcinomas of the endometrium: role for proteins involved in resistance to apoptosis and hypoxia.The WWP1 ubiquitin E3 ligase increases TRAIL resistance in breast cancer.Procaspase 8 overexpression in non-small-cell lung cancer promotes apoptosis induced by FLIP silencing.CK2 controls TRAIL and Fas sensitivity by regulating FLIP levels in endometrial carcinoma cells.Human Rhinovirus 3C protease cleaves RIPK1, concurrent with caspase 8 activation.Promoter hypermethylation and reduced expression of RASSF1A are frequent molecular alterations of endometrial carcinoma.Inhibition of CRM1 activity sensitizes endometrial and ovarian cell lines to TRAIL-induced cell death.FGFR2 alterations in endometrial carcinoma
P2860
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P2860
FLIP is frequently expressed in endometrial carcinoma and has a role in resistance to TRAIL-induced apoptosis.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
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2005年學術文章
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name
FLIP is frequently expressed i ...... ce to TRAIL-induced apoptosis.
@en
type
label
FLIP is frequently expressed i ...... ce to TRAIL-induced apoptosis.
@en
prefLabel
FLIP is frequently expressed i ...... ce to TRAIL-induced apoptosis.
@en
P2093
P2860
P1476
FLIP is frequently expressed i ...... ce to TRAIL-induced apoptosis.
@en
P2093
David Llobet
Judit Pallares
Xavier Dolcet
Xavier Matias-Guiu
P2860
P2888
P304
P356
10.1038/LABINVEST.3700286
P577
2005-07-01T00:00:00Z
P5875
P6179
1022160720