Immunological aspects of experimental allergic encephalomyelitis and multiple sclerosis.
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Live Imaging of Immune Responses in Experimental Models of Multiple SclerosisNF-κB Pathways in the Pathogenesis of Multiple Sclerosis and the Therapeutic ImplicationsLP-BM5 virus-infected mice produce activating autoantibodies to the AMPA receptorAbsence of monocyte chemoattractant protein 1 in mice leads to decreased local macrophage recruitment and antigen-specific T helper cell type 1 immune response in experimental autoimmune encephalomyelitisNeuroinflammatory imaging biomarkers: relevance to multiple sclerosis and its therapy.Activation-induced apoptosis of autoreactive and alloreactive T lymphocytes in the target organ as a major mechanism of tolerance.Presence of oligoclonal T cells in cerebrospinal fluid of a child with multiphasic disseminated encephalomyelitis following hepatitis A virus infection.Expression of 3G11 epitope defines subpopulations of regulatory T cells with different suppressive potencyNasal tolerance induction as a potential means of immunotherapy for autoimmune diseases: implications for clinical medicine.Involvement of the choroid plexus in central nervous system inflammation.Targeting ganglioside epitope 3G11 on the surface of CD4+ T cells suppresses EAE by altering the Treg/Th17 cell balanceIDO upregulates regulatory T cells via tryptophan catabolite and suppresses encephalitogenic T cell responses in experimental autoimmune encephalomyelitisExperimental autoimmune encephalomyelitis induction in genetically B cell-deficient mice.Changes in cannabinoid CB(1) receptors in striatal and cortical regions of rats with experimental allergic encephalomyelitis, an animal model of multiple sclerosis.Peptide-based immunotherapy of autoimmunity: a path of puzzles, paradoxes and possibilities.Evidence that the Y chromosome influences autoimmune disease in male and female mice.A SELDI mass spectrometry study of experimental autoimmune encephalomyelitis: sample preparation, reproducibility, and differential protein expression patterns.Multiple sclerosis as a by-product of the failure to sustain protective autoimmunity: a paradigm shift.Oligoclonal T cell repertoire in cerebrospinal fluid of patients with inflammatory diseases of the nervous system.CSF Proteomics Identifies Specific and Shared Pathways for Multiple Sclerosis Clinical SubtypesA putative mechanism of demyelination in multiple sclerosis by a proteolytic enzyme, calpain.Unmasking of an unusual myelin basic protein epitope during the process of myelin degeneration in humans: a potential mechanism for the generation of autoantigensC10 is a novel chemokine expressed in experimental inflammatory demyelinating disorders that promotes recruitment of macrophages to the central nervous system.Breakdown of the blood-retinal barrier induced by activated T cells of nonneural specificityProteomic identification of immunoproteasome accumulation in formalin-fixed rodent spinal cords with experimental autoimmune encephalomyelitisCytosolic phospholipase A₂: physiological function and role in disease.Prevention of experimental allergic encephalomyelitis by targeting nitric oxide and peroxynitrite: implications for the treatment of multiple sclerosisIncreased calpain expression in activated glial and inflammatory cells in experimental allergic encephalomyelitisSystemic immunomodulation of autoimmune disease using MHC-derived recombinant TCR ligandsCAR/FoxP3-engineered T regulatory cells target the CNS and suppress EAE upon intranasal delivery.Liver X receptor and retinoid X receptor agonists inhibit inflammatory responses of microglia and astrocytes.Immunopathogenesis and immunotherapeutic approaches in multiple sclerosis.MP4- and MOG:35-55-induced EAE in C57BL/6 mice differentially targets brain, spinal cord and cerebellum.Multitarget selection of catalytic antibodies with β-lactamase activity using phage display.Mitoxantrone repression of astrocyte activation: relevance to multiple sclerosis.Inhibition of leukotriene B4-receptor interaction suppresses eosinophil infiltration and disease pathology in a murine model of experimental allergic encephalomyelitisUnique clinical and pathological features in HLA-DRB1*0401-restricted MBP 111-129-specific humanized TCR transgenic mice.Cytosolic phospholipase A2 alpha-deficient mice are resistant to experimental autoimmune encephalomyelitis.Peroxisome proliferator-activated receptor-alpha agonist fenofibrate regulates IL-12 family cytokine expression in the CNS: relevance to multiple sclerosis.Peroxisome proliferator-activated receptor-gamma agonists suppress the production of IL-12 family cytokines by activated glia.
P2860
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P2860
Immunological aspects of experimental allergic encephalomyelitis and multiple sclerosis.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
@zh
1995年學術文章
@zh-hant
name
Immunological aspects of experimental allergic encephalomyelitis and multiple sclerosis.
@en
type
label
Immunological aspects of experimental allergic encephalomyelitis and multiple sclerosis.
@en
prefLabel
Immunological aspects of experimental allergic encephalomyelitis and multiple sclerosis.
@en
P2860
P1476
Immunological aspects of experimental allergic encephalomyelitis and multiple sclerosis.
@en
P2093
McFarland HF
P2860
P304
P356
10.3109/10408369509084683
P577
1995-01-01T00:00:00Z