Mechanisms of benzarone and benzbromarone-induced hepatic toxicity. - Wikidata - wikimedia - TriplyDB

Mechanisms of benzarone and benzbromarone-induced hepatic toxicity.

Mechanisms of benzarone and benzbromarone-induced hepatic toxicity.

http://www.wikidata.org/entity/Q40441130

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Drug Induced Steatohepatitis: An Uncommon Culprit of a Common Disease Predicting drug-induced hepatotoxicity using QSAR and toxicogenomics approaches.Hepatocellular Toxicity Associated with Tyrosine Kinase Inhibitors: Mitochondrial Damage and Inhibition of Glycolysis.Determination of drug toxicity using 3D spheroids constructed from an immortal human hepatocyte cell line.Prediction of interindividual differences in hepatic functions and drug sensitivity by using human iPS-derived hepatocytes.Discovery and Characterization of a Biologically Active Non-ATP-Competitive p38 MAP Kinase Inhibitor.Tumor necrosis factor-alpha potentiates the cytotoxicity of amiodarone in Hepa1c1c7 cells: roles of caspase activation and oxidative stress Drug-induced mitochondrial toxicity.Interaction with the hERG channel and cytotoxicity of amiodarone and amiodarone analogues.Lesinurad, a novel, oral compound for gout, acts to decrease serum uric acid through inhibition of urate transporters in the kidney.Current concepts of mechanisms in drug-induced hepatotoxicity.Oxidant stress, mitochondria, and cell death mechanisms in drug-induced liver injury: lessons learned from acetaminophen hepatotoxicity Hepatotoxicity and hepatic metabolism of available drugs: current problems and possible solutions in preclinical stages.Central role of mitochondria in drug-induced liver injury.In silico models for drug-induced liver injury--current status.Pharmacogenetic considerations in the treatment of gout.Current and future therapies for gout.Treatment Options for Gout.Mechanisms of hepatotoxicity associated with the monocyclic β-lactam antibiotic BAL30072.Association between N-desethylamiodarone/amiodarone ratio and amiodarone-induced thyroid dysfunction.Amiodarone-induced cirrhosis of liver: what predicts mortality?Mitochondrial biotransformation of omega-(phenoxy)alkanoic acids, 3-(phenoxy)acrylic acids, and omega-(1-methyl-1H-imidazol-2-ylthio)alkanoic acids: a prodrug strategy for targeting cytoprotective antioxidants to mitochondria.Acute hepatitis after amiodarone infusion.Biochemical mechanisms in drug-induced liver injury: certainties and doubts.Management of treatment resistant inflammation of acute on chronic tophaceous gout with anakinra Mechanisms of toxicity associated with six tyrosine kinase inhibitors in human hepatocyte cell lines.Biomarker discovery for drug-induced phospholipidosis: phenylacetylglycine to hippuric acid ratio in urine and plasma as potential markers.Hepatocellular Toxicity of Imidazole and Triazole Antimycotic Agents.Stronger Uricosuric Effects of the Novel Selective URAT1 Inhibitor UR-1102 Lowered Plasma Urate in Tufted Capuchin Monkeys to a Greater Extent than Benzbromarone.Autoimmune hepatitis following drug-induced liver injury in an elderly patient.Effect of Toxicants on Fatty Acid Metabolism in HepG2 Cells.

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Mechanisms of benzarone and benzbromarone-induced hepatic toxicity.

http://www.wikidata.org/entity/Q40441130

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2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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Mechanisms of benzarone and benzbromarone-induced hepatic toxicity.

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Mechanisms of benzarone and benzbromarone-induced hepatic toxicity.

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Mechanisms of benzarone and benzbromarone-induced hepatic toxicity.

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Anya Hänni

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Michael Török

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Paul Roberts

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Priska Kaufmann

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Rodolfo Gasser

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Stephan Krähenbühl

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P2860

Mitochondria and apoptosis

Mitochondrial control of cell death

Localization of the enzymes of ketogenesis in rat liver mitochondria.

Intracellular ATP, a switch in the decision between apoptosis and necrosis.

Mitochondrial dysfunction in the pathogenesis of necrotic and apoptotic cell death.

Hepatotoxicity due to mitochondrial dysfunction.

Mitochondrial intermembrane junctional complexes and their role in cell death.

Fulminant liver failure in association with the emetic toxin of Bacillus cereus.

Mitochondria: releasing power for life and unleashing the machineries of death.

The regulation of reactive oxygen species production during programmed cell death.

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925-935

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10.1002/HEP.20634

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