Cigarette smoke inhibits lipopolysaccharide-induced production of inflammatory cytokines by suppressing the activation of activator protein-1 in bronchial epithelial cells.
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Bronchiolar chemokine expression is different after single versus repeated cigarette smoke exposureInhibition of Toll-like receptor 2-mediated interleukin-8 production in Cystic Fibrosis airway epithelial cells via the alpha7-nicotinic acetylcholine receptorCytokine inhibition in the treatment of COPD.Cigarette smoke inhibits LPS-induced FABP5 expression by preventing c-Jun binding to the FABP5 promoterCigarette smoke activates the proto-oncogene c-src to promote airway inflammation and lung tissue destruction.Molecular processes that drive cigarette smoke-induced epithelial cell fate of the lung.Maternal smoking in pregnancy: do the effects on innate (toll-like receptor) function have implications for subsequent allergic disease?Exploitation of the nicotinic anti-inflammatory pathway for the treatment of epithelial inflammatory diseases.Bone marrow mononuclear cells up-regulate toll-like receptor expression and produce inflammatory mediators in response to cigarette smoke extractEffects of cigarette smoke on the activation of oxidative stress-related transcription factors in female A/J mouse lung.Comparison of nasal and bronchial epithelial cells obtained from patients with COPDIn vivo versus in vitro airway surface liquid nicotine levels following cigarette smoke exposureInhibition by cigarette smoke of nuclear factor-κB-dependent response to bacteria in the airway.Inflammatory and cytotoxic effects of acrolein, nicotine, acetylaldehyde and cigarette smoke extract on human nasal epithelial cells.Cigarette smoke-promoted acquisition of bacterial pathogens in the upper respiratory tract leads to enhanced inflammation in mice.Cigarette smoking and inflammation: cellular and molecular mechanisms.The tobacco smoke component, acrolein, suppresses innate macrophage responses by direct alkylation of c-Jun N-terminal kinase.Cigarette smoke inhibits macrophage sensing of Gram-negative bacteria and lipopolysaccharide: relative roles of nicotine and oxidant stress.COPD and squamous cell lung cancer: aberrant inflammation and immunity is the common link.The role of cathelicidin and defensins in pulmonary inflammatory diseases.Pulmonary epithelium, cigarette smoke, and chronic obstructive pulmonary disease.Human primary airway epithelial cells isolated from active smokers have epigenetically impaired antiviral responsesHow cigarette smoke skews immune responses to promote infection, lung disease and cancer.Innate immune receptors on neutrophils and their role in chronic lung disease.Inflammatory response of lung macrophages and epithelial cells to tobacco smoke: a literature review of ex vivo investigations.Impairment of phagocytosis of apoptotic cells and its role in chronic airway diseases.Periodontal disease, atherosclerosis, adverse pregnancy outcomes, and head-and-neck cancer.Methods to evaluate cytotoxicity and immunosuppression of combustible tobacco product preparations.Roflumilast N-oxide prevents cytokine secretion induced by cigarette smoke combined with LPS through JAK/STAT and ERK1/2 inhibition in airway epithelial cells.Transcriptional and epigenetic modulation of human rhinovirus-induced CXCL10 production by cigarette smoke.Sulforaphane inhibits de novo synthesis of IL-8 and MCP-1 in human epithelial cells generated by cigarette smoke extract.Cigarette smoke decreases innate responses of epithelial cells to rhinovirus infection.Cigarette smoke inhibits airway epithelial cell innate immune responses to bacteria.Cigarette smoke increases Toll-like receptor 4 and modifies lipopolysaccharide-mediated responses in airway epithelial cells.Impact of tobacco-smoke on key signaling pathways in the innate immune response in lung macrophages.Cigarette smoke extract modulates human beta-defensin-2 and interleukin-8 expression in human gingival epithelial cells.Development of an experimental model for assessing the effects of cigarette smoke and virus infections on inflammatory responses to bacterial antigens.Why does airway inflammation persist after the smoking stops?Glutathione peroxidase-1 protects against cigarette smoke-induced lung inflammation in mice.Cigarette smoke attenuates the production of cytokines by human plasmacytoid dendritic cells and enhances the release of IL-8 in response to TLR-9 stimulation.
P2860
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P2860
Cigarette smoke inhibits lipopolysaccharide-induced production of inflammatory cytokines by suppressing the activation of activator protein-1 in bronchial epithelial cells.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Cigarette smoke inhibits lipop ...... in bronchial epithelial cells.
@en
type
label
Cigarette smoke inhibits lipop ...... in bronchial epithelial cells.
@en
prefLabel
Cigarette smoke inhibits lipop ...... in bronchial epithelial cells.
@en
P1476
Cigarette smoke inhibits lipop ...... in bronchial epithelial cells
@en
P2093
Gary P Anderson
Martti Laan
P304
P356
10.4049/JIMMUNOL.173.6.4164
P407
P577
2004-09-01T00:00:00Z