Use of the red fluorescent protein as a marker of Kaposi's sarcoma-associated herpesvirus lytic gene expression.
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Identification of direct transcriptional targets of the Kaposi's sarcoma-associated herpesvirus Rta lytic switch protein by conditional nuclear localizationMolecular mechanism of BST2/tetherin downregulation by K5/MIR2 of Kaposi's sarcoma-associated herpesvirusOncogenes and RNA splicing of human tumor virusesMono-ubiquitylated ORF45 Mediates Association of KSHV Particles with Internal Lipid Rafts for Viral Assembly and EgressEnzyme Inhibition by Allosteric Capture of an Inactive ConformationThe ephrin receptor tyrosine kinase A2 is a cellular receptor for Kaposi's sarcoma–associated herpesvirusKSHV reactivation from latency requires Pim-1 and Pim-3 kinases to inactivate the latency-associated nuclear antigen LANANEDDylation is essential for Kaposi's sarcoma-associated herpesvirus latency and lytic reactivation and represents a novel anti-KSHV targetHsp70 Isoforms Are Essential for the Formation of Kaposi's Sarcoma-Associated Herpesvirus Replication and Transcription Compartments(±)-Japonones A and B, two pairs of new enantiomers with anti-KSHV activities from Hypericum japonicumVariable episomal silencing of a recombinant herpesvirus renders its encoded GFP an unreliable marker of infection in primary cells.Phosphorylation of eukaryotic translation initiation factor 4B (EIF4B) by open reading frame 45/p90 ribosomal S6 kinase (ORF45/RSK) signaling axis facilitates protein translation during Kaposi sarcoma-associated herpesvirus (KSHV) lytic replicationHistone deacetylase classes I and II regulate Kaposi's sarcoma-associated herpesvirus reactivation.Fluorescent tagging and cellular distribution of the Kaposi's sarcoma-associated herpesvirus ORF45 tegument protein.Systematic identification of cellular signals reactivating Kaposi sarcoma-associated herpesvirus.Viral oncogene-induced DNA damage response is activated in Kaposi sarcoma tumorigenesis.A comprehensive analysis of recruitment and transactivation potential of K-Rta and K-bZIP during reactivation of Kaposi's sarcoma-associated herpesvirus.Pre-micro RNA signatures delineate stages of endothelial cell transformation in Kaposi sarcoma.Non-human primate model of Kaposi's sarcoma-associated herpesvirus infectionNucleophosmin phosphorylation by v-cyclin-CDK6 controls KSHV latency.The RING-CH ligase K5 antagonizes restriction of KSHV and HIV-1 particle release by mediating ubiquitin-dependent endosomal degradation of tetherinAn important role for mitochondrial antiviral signaling protein in the Kaposi's sarcoma-associated herpesvirus life cycle.KSHV-induced notch components render endothelial and mural cell characteristics and cell survival.Kaposi Sarcoma Herpesvirus (KSHV) Latency-Associated Nuclear Antigen (LANA) recruits components of the MRN (Mre11-Rad50-NBS1) repair complex to modulate an innate immune signaling pathway and viral latencyEpigenetic analysis of KSHV latent and lytic genomes.KSHV and the pathogenesis of Kaposi sarcoma: listening to human biology and medicine.Increased efficiency of phorbol ester-induced lytic reactivation of Kaposi's sarcoma-associated herpesvirus during S phaseEpstein-Barr virus (EBV) Rta-mediated EBV and Kaposi's sarcoma-associated herpesvirus lytic reactivations in 293 cells.Enhancement of autophagy during lytic replication by the Kaposi's sarcoma-associated herpesvirus replication and transcription activator.An interaction between KSHV ORF57 and UIF provides mRNA-adaptor redundancy in herpesvirus intronless mRNA export.The lytic transcriptome of Kaposi's sarcoma-associated herpesvirus reveals extensive transcription of noncoding regions, including regions antisense to important genes.A viral nuclear noncoding RNA binds re-localized poly(A) binding protein and is required for late KSHV gene expression.Changes in nucleosome occupancy occur in a chromosome specific mannerKaposi's sarcoma herpesvirus microRNAs target caspase 3 and regulate apoptosis.Establishment and maintenance of Kaposi's sarcoma-associated herpesvirus latency in B cells.Kaposi's sarcoma herpesvirus microRNAs induce metabolic transformation of infected cellsKaposi's Sarcoma-Associated Herpesvirus microRNAs.Regulation of tumor necrosis factor-like weak inducer of apoptosis receptor protein (TWEAKR) expression by Kaposi's sarcoma-associated herpesvirus microRNA prevents TWEAK-induced apoptosis and inflammatory cytokine expression.Kaposi's sarcoma herpesvirus K15 protein contributes to virus-induced angiogenesis by recruiting PLCγ1 and activating NFAT1-dependent RCAN1 expression.Efficient lytic induction of Kaposi's sarcoma-associated herpesvirus (KSHV) by the anthracyclines.
P2860
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P2860
Use of the red fluorescent protein as a marker of Kaposi's sarcoma-associated herpesvirus lytic gene expression.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Use of the red fluorescent pro ...... esvirus lytic gene expression.
@en
type
label
Use of the red fluorescent pro ...... esvirus lytic gene expression.
@en
prefLabel
Use of the red fluorescent pro ...... esvirus lytic gene expression.
@en
P1433
P1476
Use of the red fluorescent pro ...... esvirus lytic gene expression.
@en
P2093
Jeffrey Vieira
Patricia M O'Hearn
P304
P356
10.1016/J.VIROL.2004.03.049
P407
P577
2004-08-01T00:00:00Z