STAT1 is essential for antimicrobial effector function but dispensable for gamma interferon production during Toxoplasma gondii infection.
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Inborn errors of human STAT1: allelic heterogeneity governs the diversity of immunological and infectious phenotypesThe CD40-autophagy pathway is needed for host protection despite IFN-Γ-dependent immunity and CD40 induces autophagy via control of P21 levelsSTAT1 Signaling in Astrocytes Is Essential for Control of Infection in the Central Nervous System.A novel role for Stat1 in phagosome acidification and natural host resistance to intracellular infection by Leishmania majorSevere traumatic hemorrhagic shock induces compromised immune barrier function of the mesenteric lymph node leading to an increase in intestinal bacterial translocationA mouse model of lethal infection for evaluating prophylactics and therapeutics against Monkeypox virus.p47 GTPases regulate Toxoplasma gondii survival in activated macrophages.CCR2-dependent intraepithelial lymphocytes mediate inflammatory gut pathology during Toxoplasma gondii infectionBehavior of Neutrophil Granulocytes during Toxoplasma gondii Infection in the Central Nervous System.Absence of mitogen-activated protein kinase family member c-Jun N-terminal kinase-2 enhances resistance to Toxoplasma gondiiSTAT1 signaling is essential for protection against Cryptococcus neoformans infection in mice.Subversion of host cellular functions by the apicomplexan parasites.Tumor necrosis factor alpha and gamma interferon, but not hemorrhage or pathogen burden, dictate levels of protective fibrin deposition during infectionCD40 restrains in vivo growth of Toxoplasma gondii independently of gamma interferonVirulence of Toxoplasma gondii is associated with distinct dendritic cell responses and reduced numbers of activated CD8+ T cellsToxoplasma gondii triggers phosphorylation and nuclear translocation of dendritic cell STAT1 while simultaneously blocking IFNγ-induced STAT1 transcriptional activityA partial form of recessive STAT1 deficiency in humans.Th2 signals induce epithelial injury in mice and are compatible with the biliary atresia phenotype.BoHV-4-Based Vector Single Heterologous Antigen Delivery Protects STAT1(-/-) Mice from Monkeypoxvirus Lethal Challenge.The Orphan Nuclear Receptor TLX Is an Enhancer of STAT1-Mediated Transcription and Immunity to Toxoplasma gondii.Manipulation of mitogen-activated protein kinase/nuclear factor-kappaB-signaling cascades during intracellular Toxoplasma gondii infection.STAT1 signaling within macrophages is required for antifungal activity against Cryptococcus neoformansRecruitment of Gr-1+ monocytes is essential for control of acute toxoplasmosisAltered signaling in systemic juvenile idiopathic arthritis monocytes.Primary immunodeficiencies and susceptibility to parasitic infections.IRG proteins: key mediators of interferon-regulated host resistance to intracellular pathogens.Emerging themes in IFN-gamma-induced macrophage immunity by the p47 and p65 GTPase families.TLR adaptor MyD88 is essential for pathogen control during oral toxoplasma gondii infection but not adaptive immunity induced by a vaccine strain of the parasite.Toxoplasma gondii TgIST co-opts host chromatin repressors dampening STAT1-dependent gene regulation and IFN-γ-mediated host defenses.CD40 and the immune response to parasitic infections.Dysregulation of macrophage signal transduction by Toxoplasma gondii: past progress and recent advances.Inducible nitric-oxide synthase expression is regulated by mitogen-activated protein kinase phosphatase-1.Insights into inflammatory bowel disease using Toxoplasma gondii as an infectious trigger.Downmodulation of Effector Functions in NK Cells upon Toxoplasma gondii Infection.Toxoplasma Effectors Targeting Host Signaling and Transcription.Advances in understanding immunity to Toxoplasma gondii.Mice lack of LRG-47 display the attenuated outcome of infection with Schistosoma japonicum.Infection-stimulated anemia results primarily from interferon gamma-dependent, signal transducer and activator of transcription 1-independent red cell loss.Toxoplasma Effector Recruits the Mi-2/NuRD Complex to Repress STAT1 Transcription and Block IFN-γ-Dependent Gene Expression.Involvement of Stat1 in the phagocytosis of M. avium.
P2860
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P2860
STAT1 is essential for antimicrobial effector function but dispensable for gamma interferon production during Toxoplasma gondii infection.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
STAT1 is essential for antimic ...... g Toxoplasma gondii infection.
@en
type
label
STAT1 is essential for antimic ...... g Toxoplasma gondii infection.
@en
prefLabel
STAT1 is essential for antimic ...... g Toxoplasma gondii infection.
@en
P2093
P2860
P1476
STAT1 is essential for antimic ...... ng Toxoplasma gondii infection
@en
P2093
Barbara A Butcher
Eric Y Denkers
Gregory A Taylor
L Cristina Gavrilescu
P2860
P304
P356
10.1128/IAI.72.3.1257-1264.2004
P407
P577
2004-03-01T00:00:00Z