Apolipoprotein B100 exit from the endoplasmic reticulum (ER) is COPII-dependent, and its lipidation to very low density lipoprotein occurs post-ER.
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Molecular processes that handle -- and mishandle -- dietary lipidsMechanisms of lipid malabsorption in Cystic Fibrosis: the impact of essential fatty acids deficiencyVesicle-mediated ER export of proteins and lipidsHepatitis C virus production by human hepatocytes dependent on assembly and secretion of very low-density lipoproteinsCellular Determinants of Hepatitis C Virus Assembly, Maturation, Degradation, and SecretionFunctional analysis of the missense APOC3 mutation Ala23Thr associated with human hypotriglyceridemiaThe identification of a novel endoplasmic reticulum to Golgi SNARE complex used by the prechylomicron transport vesicleGolgi-associated maturation of very low density lipoproteins involves conformational changes in apolipoprotein B, but is not dependent on apolipoprotein EMolecular analysis and intestinal expression of SAR1 genes and proteins in Anderson's disease (Chylomicron retention disease)Self-association and lipid binding properties of the lipoprotein initiating domain of apolipoprotein B.Recent progress in understanding protein and lipid factors affecting hepatic VLDL assembly and secretion.Control of very low-density lipoprotein secretion by N-ethylmaleimide-sensitive factor and miR-33ACAT2 stimulates cholesteryl ester secretion in apoB-containing lipoproteins.Lpcat3-dependent production of arachidonoyl phospholipids is a key determinant of triglyceride secretionRat carboxylesterase ES-4 enzyme functions as a major hepatic neutral cholesteryl ester hydrolase.Mature VLDL triggers the biogenesis of a distinct vesicle from the trans-Golgi network for its export to the plasma membrane.Hepatic ABCA1 and VLDL triglyceride production.The [corrected] SEC23-SEC31 [corrected] interface plays critical role for export of procollagen from the endoplasmic reticulumIntracellular trafficking and secretion of VLDL.Proteomic analysis of the very low density lipoprotein (VLDL) transport vesicles.Effect of Octreotide on Hepatic Steatosis in Diet-Induced Obesity in Rats.Opposing roles of cell death-inducing DFF45-like effector B and perilipin 2 in controlling hepatic VLDL lipidation.Presecretory oxidation, aggregation, and autophagic destruction of apoprotein-B: a pathway for late-stage quality controlCideB protein is required for the biogenesis of very low density lipoprotein (VLDL) transport vesicleRole of the gut in lipid homeostasisStructural and dynamic interfacial properties of the lipoprotein initiating domain of apolipoprotein B.Silencing of Small Valosin-containing Protein-interacting Protein (SVIP) Reduces Very Low Density Lipoprotein (VLDL) Secretion from Rat Hepatocytes by Disrupting Its Endoplasmic Reticulum (ER)-to-Golgi TraffickingReconstituting initial events during the assembly of apolipoprotein B-containing lipoproteins in a cell-free system.The many intersecting pathways underlying apolipoprotein B secretion and degradationGlycosyltransferase GLT8D2 positively regulates ApoB100 protein expression in hepatocytes.Mea6 controls VLDL transport through the coordinated regulation of COPII assembly.A regulator of secretory vesicle size, Kelch-like protein 12, facilitates the secretion of apolipoprotein B100 and very-low-density lipoproteins--brief report.Docosahexaenoic acid impairs the maturation of very low density lipoproteins in rat hepatic cells.Apolipoprotein B100 biogenesis: a complex array of intracellular mechanisms regulating folding, stability, and lipoprotein assembly.Virion assembly and release.The pathophysiology of intestinal lipoprotein production.Hepatic Tm6sf2 overexpression affects cellular ApoB-trafficking, plasma lipid levels, hepatic steatosis and atherosclerosis.Hypoxia-inducible lipid droplet-associated (HILPDA) is a novel peroxisome proliferator-activated receptor (PPAR) target involved in hepatic triglyceride secretion.Hrd1 participates in the regulation of collagen I synthesis in renal fibrosis.Ultrastructural and biochemical basis for hepatitis C virus morphogenesis.
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P2860
Apolipoprotein B100 exit from the endoplasmic reticulum (ER) is COPII-dependent, and its lipidation to very low density lipoprotein occurs post-ER.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Apolipoprotein B100 exit from ...... ty lipoprotein occurs post-ER.
@en
type
label
Apolipoprotein B100 exit from ...... ty lipoprotein occurs post-ER.
@en
prefLabel
Apolipoprotein B100 exit from ...... ty lipoprotein occurs post-ER.
@en
P2093
P2860
P356
P1476
Apolipoprotein B100 exit from ...... ty lipoprotein occurs post-ER.
@en
P2093
Edward A Fisher
Jeffrey L Brodsky
Viktoria Gusarova
P2860
P304
48051-48058
P356
10.1074/JBC.M306898200
P407
P577
2003-09-05T00:00:00Z