Induction of in vitro reprogramming by Toll-like receptor (TLR)2 and TLR4 agonists in murine macrophages: effects of TLR "homotolerance" versus "heterotolerance" on NF-kappa B signaling pathway components.
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Zinc finger protein Gfi1 controls the endotoxin-mediated Toll-like receptor inflammatory response by antagonizing NF-kappaB p65Endotoxin tolerance impairs IL-1 receptor-associated kinase (IRAK) 4 and TGF-beta-activated kinase 1 activation, K63-linked polyubiquitination and assembly of IRAK1, TNF receptor-associated factor 6, and IkappaB kinase gamma and increases A20 expresHCV-induced miR-21 contributes to evasion of host immune system by targeting MyD88 and IRAK1Immunosenescence and macrophage functional plasticity: dysregulation of macrophage function by age-associated microenvironmental changesAirway epithelial cell tolerance to Pseudomonas aeruginosa.MicroRNA-21 in the pathogenesis of acute kidney injuryTolerance and Cross-Tolerance following Toll-Like Receptor (TLR)-4 and -9 Activation Are Mediated by IRAK-M and Modulated by IL-7 in Murine SplenocytesChronic filarial infection provides protection against bacterial sepsis by functionally reprogramming macrophagesViral and Host Factors Induce Macrophage Activation and Loss of Toll-Like Receptor Tolerance in Chronic HCV InfectionSpace and time: New considerations about the relationship between Toll-like receptors (TLRs) and type I interferons (IFNs)Persistently active microbial molecules prolong innate immune tolerance in vivoThe involvement of cholesterol in sepsis and tolerance to lipopolysaccharide highlighted by the transcriptome analysis of zebrafish (Danio rerio)LPS-induced TNF-alpha factor (LITAF)-deficient mice express reduced LPS-induced cytokine: Evidence for LITAF-dependent LPS signaling pathwaysToll-like receptors and their adaptors are regulated in macrophages after phagocytosis of lipopolysaccharide-coated titanium particlesZinc finger protein 64 promotes Toll-like receptor-triggered proinflammatory and type I interferon production in macrophages by enhancing p65 subunit activationCharacterization of a Plasmodium falciparum macrophage-migration inhibitory factor homologueSustained desensitization to bacterial Toll-like receptor ligands after resolution of respiratory influenza infection.Sex effects on neurodevelopmental outcomes of innate immune activation during prenatal and neonatal life.Transcriptome analysis of Litopenaeus vannamei in response to white spot syndrome virus infectionGenome wide transcriptome analysis of dendritic cells identifies genes with altered expression in psoriasis.Tolerance induced via TLR2 and TLR4 in human dendritic cells: role of IRAK-1.The role of NF-kappaB and H3K27me3 demethylase, Jmjd3, on the anthrax lethal toxin tolerance of RAW 264.7 cells.Leishmania donovani isolates with antimony-resistant but not -sensitive phenotype inhibit sodium antimony gluconate-induced dendritic cell activation.The potential of p38 MAPK inhibitors to modulate periodontal infections.Pathophysiology of endotoxin tolerance: mechanisms and clinical consequences.Oral mucosal endotoxin tolerance induction in chronic periodontitis.Calcineurin inactivation leads to decreased responsiveness to LPS in macrophages and dendritic cells and protects against LPS-induced toxicity in vivoDevelopmentally regulated IkappaB expression in intestinal epithelium and susceptibility to flagellin-induced inflammationPeriodontal innate immune mechanisms relevant to atherosclerosis and obesity.Effects of aging on endotoxin tolerance induced by lipopolysaccharides derived from Porphyromonas gingivalis and Escherichia coli.IL-10-dependent partial refractoriness to Toll-like receptor stimulation modulates gut mucosal dendritic cell function.Co-infection of helminths and malaria: modulation of the immune responses to malaria.Toll-like receptor 9: a new target of ischemic preconditioning in the brain.Human endometrial epithelial cells cyclically express Toll-like receptor 3 (TLR3) and exhibit TLR3-dependent responses to dsRNA.MicroRNA-146a is upregulated by and negatively regulates TLR2 signaling.Reprogramming of murine macrophages through TLR2 confers viral resistance via TRAF3-mediated, enhanced interferon production.Host inactivation of bacterial lipopolysaccharide prevents prolonged tolerance following gram-negative bacterial infection.Macrophage subset sensitivity to endotoxin tolerisation by Porphyromonas gingivalisPorphyromonas gingivalis lipopolysaccharide antagonizes Escherichia coli lipopolysaccharide at toll-like receptor 4 in human endothelial cells.Periodontal innate immune mechanisms relevant to atherosclerosis.
P2860
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P2860
Induction of in vitro reprogramming by Toll-like receptor (TLR)2 and TLR4 agonists in murine macrophages: effects of TLR "homotolerance" versus "heterotolerance" on NF-kappa B signaling pathway components.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Induction of in vitro reprogra ...... signaling pathway components.
@en
type
label
Induction of in vitro reprogra ...... signaling pathway components.
@en
prefLabel
Induction of in vitro reprogra ...... signaling pathway components.
@en
P2093
P1476
Induction of in vitro reprogra ...... signaling pathway components.
@en
P2093
Andrei E Medvedev
Karen E Thomas
Marina A Dobrovolskaia
Michael J Cody
Nancy R Rice
Natalia Cuesta
Stefanie N Vogel
Suzanne M Michalek
Tianbo Ren
Vladimir Toshchakov
P304
P356
10.4049/JIMMUNOL.170.1.508
P407
P577
2003-01-01T00:00:00Z