PI3K/AKT signaling regulates bioenergetics in immortalized hepatocytes. - Wikidata - wikimedia - TriplyDB

PI3K/AKT signaling regulates bioenergetics in immortalized hepatocytes.

PI3K/AKT signaling regulates bioenergetics in immortalized hepatocytes.

http://www.wikidata.org/entity/Q40705320

about

Adipose tissue insulin resistance due to loss of PI3K p110α leads to decreased energy expenditure and obesity A novel pharmacological strategy by PTEN inhibition for improving metabolic resuscitation and survival after mouse cardiac arrest.Insulin resistance is associated with epigenetic and genetic regulation of mitochondrial DNA in obese humans.Adaptive mitochondrial reprogramming and resistance to PI3K therapy.Twist promotes reprogramming of glucose metabolism in breast cancer cells through PI3K/AKT and p53 signaling pathways.Akt phosphorylates HK-II at Thr-473 and increases mitochondrial HK-II association to protect cardiomyocytes Synaptic activity and bioenergy homeostasis: implications in brain trauma and neurodegenerative diseases.Mitochondrial function in ageing: coordination with signalling and transcriptional pathways.Akt2-Dependent Phosphorylation of Radixin in Regulation of Mrp-2 Trafficking in WIF-B Cells.Regulation of O2 consumption by the PI3K and mTOR pathways contributes to tumor hypoxia.AKT-mediated phosphorylation of ATG4B impairs mitochondrial activity and enhances the Warburg effect in hepatocellular carcinoma cells.Crosstalk of LKB1- and PTEN-regulated signals in liver morphogenesis and tumor development.Phosphoinositide 3-Kinase/Akt Signaling and Redox Metabolism in Cancer.RNS60 exerts therapeutic effects in the SOD1 ALS mouse model through protective glia and peripheral nerve rescue.Brain metabolic and functional alterations in a liver-specific PTEN knockout mouse model

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P2860

PI3K/AKT signaling regulates bioenergetics in immortalized hepatocytes.

http://www.wikidata.org/entity/Q40705320

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

PI3K/AKT signaling regulates bioenergetics in immortalized hepatocytes.

@en

type

label

PI3K/AKT signaling regulates bioenergetics in immortalized hepatocytes.

@en

prefLabel

PI3K/AKT signaling regulates bioenergetics in immortalized hepatocytes.

@en

P1433

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P356

J.FREERADBIOMED.2013.01.013

P1433

Free Radical Biology and Medicine

P1476

PI3K/AKT signaling regulates bioenergetics in immortalized hepatocytes.

@en

P2093

Amit R Agarwal

http://www.w3.org/2001/XMLSchema#string

Bangyan L Stiles

http://www.w3.org/2001/XMLSchema#string

Chen Li

http://www.w3.org/2001/XMLSchema#string

Enrique Cadenas

http://www.w3.org/2001/XMLSchema#string

Lina He

http://www.w3.org/2001/XMLSchema#string

Ni Zeng

http://www.w3.org/2001/XMLSchema#string

Yang Li

http://www.w3.org/2001/XMLSchema#string

P2860

Akt, a pleckstrin homology domain containing kinase, is activated primarily by phosphorylation

The tumor suppressor, PTEN/MMAC1, dephosphorylates the lipid second messenger, phosphatidylinositol 3,4,5-trisphosphate

Understanding the Warburg effect: the metabolic requirements of cell proliferation

Akt1 intramitochondrial cycling is a crucial step in the redox modulation of cell cycle progression

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

The phosphoinositide 3-kinase pathway

Type I phosphatidylinositol kinase makes a novel inositol phospholipid, phosphatidylinositol-3-phosphate

Inhibition of early apoptotic events by Akt/PKB is dependent on the first committed step of glycolysis and mitochondrial hexokinase

Hexokinase-mitochondria interaction mediated by Akt is required to inhibit apoptosis in the presence or absence of Bax and Bak

Signaling by phosphoinositide-3,4,5-trisphosphate through proteins containing pleckstrin and Sec7 homology domains

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29-40

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scholarly article

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10.1016/J.FREERADBIOMED.2013.01.013

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60

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2013-01-30T00:00:00Z

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235396727

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23376468

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3654039

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