Murine coronavirus delays expression of a subset of interferon-stimulated genes
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PLP2 of mouse hepatitis virus A59 (MHV-A59) targets TBK1 to negatively regulate cellular type I interferon signaling pathway.PKR mediated regulation of inflammation and IL-10 during viral encephalomyelitis.Coronavirus papain-like proteases negatively regulate antiviral innate immune response through disruption of STING-mediated signalingAutocrine interferon priming in macrophages but not dendritic cells results in enhanced cytokine and chemokine production after coronavirus infectionMERS-coronavirus replication induces severe in vitro cytopathology and is strongly inhibited by cyclosporin A or interferon-α treatmentAnalysis of the host transcriptome from demyelinating spinal cord of murine coronavirus-infected mice.Ribose 2'-O-methylation provides a molecular signature for the distinction of self and non-self mRNA dependent on the RNA sensor Mda5.Cell-type-specific type I interferon antagonism influences organ tropism of murine coronavirusThe nsp1, nsp13, and M proteins contribute to the hepatotropism of murine coronavirus JHM.WU.Recent progress in studies of arterivirus- and coronavirus-host interactionsD471G mutation in LCMV-NP affects its ability to self-associate and results in a dominant negative effect in viral RNA synthesisCell-type-specific activation of the oligoadenylate synthetase-RNase L pathway by a murine coronavirus.Identification of an infectious bronchitis coronavirus strain exhibiting a classical genotype but altered antigenicity, pathogenicity, and innate immunity profile.Activation of the chicken type I interferon response by infectious bronchitis coronavirus.Rescue of recombinant Newcastle disease virus from cDNA.Endoplasmic reticulum stress regulates the innate immunity critical transcription factor IRF3.Simian varicella virus inhibits the interferon gamma signalling pathway.Extracellular matrix in the CNS induced by neuropathogenic viral infection.Infectious Bronchitis Coronavirus Inhibits STAT1 Signaling and Requires Accessory Proteins for Resistance to Type I Interferon Activity.Antagonism of the interferon-induced OAS-RNase L pathway by murine coronavirus ns2 protein is required for virus replication and liver pathology.Mice lacking α1,3-fucosyltransferase 9 exhibit modulation of in vivo immune responses against pathogens.Murine hepatitis virus nsp14 exoribonuclease activity is required for resistance to innate immunity.
P2860
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P2860
Murine coronavirus delays expression of a subset of interferon-stimulated genes
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Murine coronavirus delays expression of a subset of interferon-stimulated genes
@en
type
label
Murine coronavirus delays expression of a subset of interferon-stimulated genes
@en
prefLabel
Murine coronavirus delays expression of a subset of interferon-stimulated genes
@en
P2860
P50
P356
P1433
P1476
Murine coronavirus delays expression of a subset of interferon-stimulated genes
@en
P2093
Kristine M Rose
P2860
P304
P356
10.1128/JVI.00211-10
P407
P577
2010-03-31T00:00:00Z