Too much of a good thing: Sustained type 1 interferon signaling limits humoral responses to secondary viral infection.

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Too much of a good thing: Sustained type 1 interferon signaling limits humoral responses to secondary viral infection.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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2016年學術文章

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Too much of a good thing: Sust ...... to secondary viral infection.

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Too much of a good thing: Sust ...... to secondary viral infection.

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Too much of a good thing: Sust ...... to secondary viral infection.

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P1476

Too much of a good thing: Sust ...... to secondary viral infection.

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P2093

John R Teijaro

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Interleukin-10 determines viral clearance or persistence in vivo

Restoring function in exhausted CD8 T cells during chronic viral infection

General and specific immunosuppression caused by antiviral T-cell responses.

Persistent virus infection inhibits type I interferon production by plasmacytoid dendritic cells to facilitate opportunistic infections.

Absence of mouse 2B4 promotes NK cell-mediated killing of activated CD8+ T cells, leading to prolonged viral persistence and altered pathogenesis.

Virus-induced immunosuppression: immune system-mediated destruction of virus-infected dendritic cells results in generalized immune suppression.

Emergence of distinct multiarmed immunoregulatory antigen-presenting cells during persistent viral infection

Viral targeting of fibroblastic reticular cells contributes to immunosuppression and persistence during chronic infection.

Infected CD8α- dendritic cells are the predominant source of IL-10 during establishment of persistent viral infection.

Natural killer cells act as rheostats modulating antiviral T cells.

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300-302

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scholarly article

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10.1002/EJI.201546224

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English

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2016-01-18T00:00:00Z

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26783074

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5113021

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