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Atypical PKCs in memory maintenance: the roles of feedback and redundancy

Atypical PKCs in memory maintenance: the roles of feedback and redundancy

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Environmental Enrichment Increases Glucocorticoid Receptors and Decreases GluA2 and Protein Kinase M Zeta (PKMζ) Trafficking During Chronic Stress: A Protective Mechanism?Persistent Associative Plasticity at an Identified Synapse Underlying Classical Conditioning Becomes Labile with Short-Term Homosynaptic Activation.Memory Takes Time.Computational principles of memory.Cell-Specific PKM Isoforms Contribute to the Maintenance of Different Forms of Persistent Long-Term Synaptic Plasticity.The Contribution of Spatial and Temporal Molecular Networks in the Induction of Long-term Memory and Its Underlying Synaptic Plasticity Persistent increased PKMζ in long-term and remote spatial memory.Intracerebellar infusion of the protein kinase M zeta (PKMζ) inhibitor zeta-inhibitory peptide (ZIP) disrupts eyeblink classical conditioning.Criteria for identifying the molecular basis of the engram (CaMKII, PKMzeta).Selective Erasure of Distinct Forms of Long-Term Synaptic Plasticity Underlying Different Forms of Memory in the Same Postsynaptic Neuron.Memory Synapses Are Defined by Distinct Molecular Complexes: A Proposal.Coupled feedback loops maintain synaptic long-term potentiation: A computational model of PKMzeta synthesis and AMPA receptor trafficking.The Synaptic Theory of Memory: A Historical Survey and Reconciliation of Recent Opposition

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P2860

Atypical PKCs in memory maintenance: the roles of feedback and redundancy

http://www.wikidata.org/entity/Q40832660

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2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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2015年學術文章

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2015年學術文章

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Atypical PKCs in memory maintenance: the roles of feedback and redundancy

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Atypical PKCs in memory maintenance: the roles of feedback and redundancy

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Atypical PKCs in memory maintenance: the roles of feedback and redundancy

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Learning and Memory

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Atypical PKCs in memory maintenance: the roles of feedback and redundancy

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Harel Z Shouval

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Sajiya J Jalil

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Todd Charlton Sacktor

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P2860

PKMzeta maintains spatial, instrumental, and classically conditioned long-term memories

Activation of PKCzeta and PKMzeta in the nucleus accumbens core is necessary for the retrieval, consolidation and reconsolidation of drug memory

A synaptic model of memory: long-term potentiation in the hippocampus

mTORC2 phosphorylates protein kinase Cζ to regulate its stability and activity

Essential function of TORC2 in PKC and Akt turn motif phosphorylation, maturation and signalling

Long-lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant path

Persistent activation of the zeta isoform of protein kinase C in the maintenance of long-term potentiation

Homosynaptic long-term depression in area CA1 of hippocampus and effects of N-methyl-D-aspartate receptor blockade

PKM-ζ is not required for hippocampal synaptic plasticity, learning and memory

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344-353

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scholarly article

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10.1101/LM.038844.115

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7

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22

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4478332

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