The cellular and molecular pathogenesis of coronaviruses.
about
A double epidemic model for the SARS propagationTargeted disruption of the Ceacam1 (MHVR) gene leads to reduced susceptibility of mice to mouse hepatitis virus infectionRNase L-independent specific 28S rRNA cleavage in murine coronavirus-infected cellsNeuroinvasion by human respiratory coronaviruses.Pathogenesis of chimeric MHV4/MHV-A59 recombinant viruses: the murine coronavirus spike protein is a major determinant of neurovirulence.Induction of apoptosis in murine coronavirus-infected cultured cells and demonstration of E protein as an apoptosis inducer.Purified, soluble recombinant mouse hepatitis virus receptor, Bgp1(b), and Bgp2 murine coronavirus receptors differ in mouse hepatitis virus binding and neutralizing activitiesMurine coronavirus-induced subacute fatal peritonitis in C57BL/6 mice deficient in gamma interferon.Comparisons of the F and HN gene sequences of different strains of bovine parainfluenza virus type 3: relationship to phenotype and pathogenicity.Murine coronavirus replication induces cell cycle arrest in G0/G1 phase.Both spike and background genes contribute to murine coronavirus neurovirulenceA hypervariable region within the 3' cis-acting element of the murine coronavirus genome is nonessential for RNA synthesis but affects pathogenesisMouse hepatitis virus-specific cytotoxic T lymphocytes protect from lethal infection without eliminating virus from the central nervous system.Analysis of second-site revertants of a murine coronavirus nucleocapsid protein deletion mutant and construction of nucleocapsid protein mutants by targeted RNA recombination.Mouse hepatitis virus is cleared from the central nervous systems of mice lacking perforin-mediated cytolysis.The internal open reading frame within the nucleocapsid gene of mouse hepatitis virus encodes a structural protein that is not essential for viral replicationThe challenges of implementing pathogen control strategies for fishes used in biomedical research.Characterization of the Ld-restricted cytotoxic T-lymphocyte epitope in the mouse hepatitis virus nucleocapsid protein.Mouse hepatitis virus infection of the CNS: a model for defense, disease, and repair.Murine coronavirus nonstructural protein p28 arrests cell cycle in G0/G1 phase.Senescent BALB/c mice are able to develop resistance to Leishmania major infection.The viral spike protein is not involved in the polarized sorting of coronaviruses in epithelial cells.The ubiquitin-proteasome system plays an important role during various stages of the coronavirus infection cycle.Spread of a neurotropic coronavirus to spinal cord white matter via neurons and astrocytes.Cytokine induction during T-cell-mediated clearance of mouse hepatitis virus from neurons in vivo.Murine Olfactory Bulb Interneurons Survive Infection with a Neurotropic Coronavirus.Receptor-dependent coronavirus infection of dendritic cells.Coronavirus infections in the laboratory rat: degree of cross protection following immunization with a heterologous strain.Interference of natural mouse hepatitis virus infection with cytokine production and susceptibility to Trypanosoma cruzi.
P2860
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P2860
The cellular and molecular pathogenesis of coronaviruses.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
The cellular and molecular pathogenesis of coronaviruses.
@en
type
label
The cellular and molecular pathogenesis of coronaviruses.
@en
prefLabel
The cellular and molecular pathogenesis of coronaviruses.
@en
P2093
P1433
P1476
The cellular and molecular pathogenesis of coronaviruses
@en
P2093
P577
1993-02-01T00:00:00Z