Mouse hepatitis virus-specific cytotoxic T lymphocytes protect from lethal infection without eliminating virus from the central nervous system.
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Fas Ligand Interactions Contribute to CD8+ T-Cell-Mediated Control of West Nile Virus Infection in the Central Nervous SystemCharacterization of murine coronavirus neutralization epitopes with phage-displayed peptidesIncreased epitope-specific CD8+ T cells prevent murine coronavirus spread to the spinal cord and subsequent demyelination.Receptor-independent spread of a highly neurotropic murine coronavirus JHMV strain from initially infected microglial cells in mixed neural culturesContributions of Fas-Fas ligand interactions to the pathogenesis of mouse hepatitis virus in the central nervous system.Role of viral persistence in retaining CD8(+) T cells within the central nervous systemVirus-neutralizing monoclonal antibody expressed in milk of transgenic mice provides full protection against virus-induced encephalitis.A mechanism of virus-induced demyelination.Pathogenesis of murine coronavirus in the central nervous system.Matrix metalloproteinase expression correlates with virulence following neurotropic mouse hepatitis virus infectionPreferential infection of mature dendritic cells by mouse hepatitis virus strain JHMKinetics of virus-specific CD8+ -T-cell expansion and trafficking following central nervous system infection.Both spike and background genes contribute to murine coronavirus neurovirulenceControl of central nervous system viral persistence by neutralizing antibody.Effects of an epitope-specific CD8+ T-cell response on murine coronavirus central nervous system disease: protection from virus replication and antigen spread and selection of epitope escape mutants.In vivo expression of major histocompatibility complex molecules on oligodendrocytes and neurons during viral infectionCD8+ T-cell epitopes within the surface glycoprotein of a neurotropic coronavirus and correlation with pathogenicity.Specificity of the H-2 L(d)-restricted cytotoxic T-lymphocyte response to the mouse hepatitis virus nucleocapsid proteinMouse hepatitis virus is cleared from the central nervous systems of mice lacking perforin-mediated cytolysis.Antiviral CD8 T cells recognize borna disease virus antigen transgenically expressed in either neurons or astrocytesPriming of CD8+ T cells during central nervous system infection with a murine coronavirus is strain dependent.Perforin and gamma interferon-mediated control of coronavirus central nervous system infection by CD8 T cells in the absence of CD4 T cellsRegulation of proinflammatory cytokine expression in primary mouse astrocytes by coronavirus infectionTumor necrosis factor expression during mouse hepatitis virus-induced demyelinating encephalomyelitis.The carboxyl-terminal 120-residue polypeptide of infectious bronchitis virus nucleocapsid induces cytotoxic T lymphocytes and protects chickens from acute infection.Activated GL7+ B cells are maintained within the inflamed CNS in the absence of follicle formation during viral encephalomyelitis.S protein of severe acute respiratory syndrome-associated coronavirus mediates entry into hepatoma cell lines and is targeted by neutralizing antibodies in infected patients.
P2860
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P2860
Mouse hepatitis virus-specific cytotoxic T lymphocytes protect from lethal infection without eliminating virus from the central nervous system.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Mouse hepatitis virus-specific ...... om the central nervous system.
@ast
Mouse hepatitis virus-specific ...... om the central nervous system.
@en
type
label
Mouse hepatitis virus-specific ...... om the central nervous system.
@ast
Mouse hepatitis virus-specific ...... om the central nervous system.
@en
prefLabel
Mouse hepatitis virus-specific ...... om the central nervous system.
@ast
Mouse hepatitis virus-specific ...... om the central nervous system.
@en
P2093
P2860
P1433
P1476
Mouse hepatitis virus-specific ...... om the central nervous system.
@en
P2093
C C Bergmann
D R Hinton
R C van der Veen
S A Stohlman
P2860
P304
P407
P577
1995-02-01T00:00:00Z