A systemic defect in Toll-like receptor 4 signaling increases lipopolysaccharide-induced suppression of IL-2-dependent T-cell proliferation in COPD.
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B cells in chronic obstructive pulmonary disease: moving to center stage.Pulmonary Epithelial TLR4 Activation Leads to Lung Injury in Neonatal Necrotizing Enterocolitis.LPS enhances TLR4 expression and IFN‑γ production via the TLR4/IRAK/NF‑κB signaling pathway in rat pulmonary arterial smooth muscle cells.The value of blood cytokines and chemokines in assessing COPD.IRAK Inhibitor Protects the Intestinal Tract of Necrotizing Enterocolitis by Inhibiting the Toll-Like Receptor (TLR) Inflammatory Signaling Pathway in Rats.
P2860
A systemic defect in Toll-like receptor 4 signaling increases lipopolysaccharide-induced suppression of IL-2-dependent T-cell proliferation in COPD.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
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2015年论文
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2015年论文
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name
A systemic defect in Toll-like ...... T-cell proliferation in COPD.
@en
type
label
A systemic defect in Toll-like ...... T-cell proliferation in COPD.
@en
prefLabel
A systemic defect in Toll-like ...... T-cell proliferation in COPD.
@en
P2093
P2860
P1476
A systemic defect in Toll-like ...... t T-cell proliferation in COPD
@en
P2093
Andrea Koch
David Jungck
Sarah Yanik
Sarah-Jane Chikosi
P2860
P304
P356
10.1152/AJPLUNG.00367.2014
P577
2015-10-23T00:00:00Z