Physicochemical and histological changes in the arterial wall of nonhuman primates during progression and regression of atherosclerosis.
about
Cholesterol lowering and coronary artery disease: mechanisms of risk reduction.Regression of atherosclerosis--an ounce of prevention.Why are low-density lipoproteins atherogenic?Arteriographic view of treatment to achieve regression of coronary atherosclerosis and to prevent plaque disruption and clinical cardiovascular events.Phase behavior and crystalline structures of cholesteryl ester mixtures: a C-13 MASNMR study.The potential role of AT(1)-receptor blockade in the prevention and reversal of atherosclerosis.Suppressed monocyte recruitment drives macrophage removal from atherosclerotic plaques of Apoe-/- mice during disease regressionHistological changes and risk factor associations in type 2 atherosclerotic lesions (fatty streaks) in young adults.Homozygosity for an allele encoding deacetylated FoxO1 protects macrophages from cholesterol-induced inflammation without increasing apoptosis.Celery Seed Extract Blocks Peroxide Injury in Macrophages via Notch1/NF-κB Pathway.Niemann-Pick C heterozygosity confers resistance to lesional necrosis and macrophage apoptosis in murine atherosclerosis.Cholesterol-induced macrophage apoptosis requires ER stress pathways and engagement of the type A scavenger receptor.Prospects for atherosclerosis regression through increase in high-density lipoprotein and other emerging therapeutic targetsUtilizing imaging tools in lipidology: examining the potential of MRI for monitoring cholesterol therapy.Overexpression of transforming growth factor beta1 in arterial endothelium causes hyperplasia, apoptosis, and cartilaginous metaplasia.Lymphatic vasculature mediates macrophage reverse cholesterol transport in miceMR imaging of carotid plaque composition during lipid-lowering therapy a prospective assessment of effect and time course.Chronic miR-29 antagonism promotes favorable plaque remodeling in atherosclerotic miceIL-22 is induced by S100/calgranulin and impairs cholesterol efflux in macrophages by downregulating ABCG1Atherosclerosis regression and high-density lipoproteins.Targeting cholesterol crystal-induced inflammation for the secondary prevention of cardiovascular disease.ABCA1-mediated cholesterol efflux is defective in free cholesterol-loaded macrophages. Mechanism involves enhanced ABCA1 degradation in a process requiring full NPC1 activity.Concentration-Dependent Diversifcation Effects of Free Cholesterol Loading on Macrophage Viability and Polarization.Enrichment of endoplasmic reticulum with cholesterol inhibits sarcoplasmic-endoplasmic reticulum calcium ATPase-2b activity in parallel with increased order of membrane lipids: implications for depletion of endoplasmic reticulum calcium stores and aAtherosclerosis: what is it and why does it occur?Spontaneous formation of two-dimensional and three-dimensional cholesterol crystals in single hydrated lipid bilayers.Targeted Knockdown of Hepatic SOAT2 With Antisense Oligonucleotides Stabilizes Atherosclerotic Plaque in ApoB100-only LDLr-/- Mice.Endoplasmic reticulum stress-related inflammation and cardiovascular diseases.Risk of thrombosis in human atherosclerotic plaques: role of extracellular lipid, macrophage, and smooth muscle cell content.Acid sphingomyelinase-deficient macrophages have defective cholesterol trafficking and efflux.Apoptosis and plaque destabilization in atherosclerosis: the role of macrophage apoptosis induced by cholesterol.Free cholesterol-loaded macrophages are an abundant source of tumor necrosis factor-alpha and interleukin-6: model of NF-kappaB- and map kinase-dependent inflammation in advanced atherosclerosis.Cholesterol-induced apoptotic macrophages elicit an inflammatory response in phagocytes, which is partially attenuated by the Mer receptor.Cholesterol accumulation inhibits ER to Golgi transport and protein secretion: studies of apolipoprotein E and VSVGt.Reversal of gangrenous lesions in the blue toe syndrome with lovastatin--a case report.Macrophages deficient in CTP:Phosphocholine cytidylyltransferase-alpha are viable under normal culture conditions but are highly susceptible to free cholesterol-induced death. Molecular genetic evidence that the induction of phosphatidylcholine biosCarotid thin fluttering bands: A new element of arterial wall remodelling? An ultrasound study.
P2860
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P2860
Physicochemical and histological changes in the arterial wall of nonhuman primates during progression and regression of atherosclerosis.
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
Physicochemical and histologic ...... regression of atherosclerosis.
@en
type
label
Physicochemical and histologic ...... regression of atherosclerosis.
@en
prefLabel
Physicochemical and histologic ...... regression of atherosclerosis.
@en
P2093
P2860
P356
P1476
Physicochemical and histologic ...... regression of atherosclerosis.
@en
P2093
P2860
P304
P356
10.1172/JCI111366
P407
P577
1984-06-01T00:00:00Z