Convallaria keiskei as a novel therapeutic alternative for salivary gland cancer treatment by targeting myeloid cell leukemia-1.

about

Convallaria keiskei as a novel therapeutic alternative for salivary gland cancer treatment by targeting myeloid cell leukemia-1.

Item

http://www.wikidata.org/entity/Q41011587

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Convallaria keiskei as a novel ...... eting myeloid cell leukemia-1.

@en

type

Item

label

Convallaria keiskei as a novel ...... eting myeloid cell leukemia-1.

@en

prefLabel

Convallaria keiskei as a novel ...... eting myeloid cell leukemia-1.

@en

P1476

Convallaria keiskei as a novel ...... eting myeloid cell leukemia-1.

@en

P2093

Haeng-Eun Lee

http://www.w3.org/2001/XMLSchema#string

In-Hyoung Yang

http://www.w3.org/2001/XMLSchema#string

In-Sun Hong

http://www.w3.org/2001/XMLSchema#string

Jeong-Seok Nam

http://www.w3.org/2001/XMLSchema#string

Ji-Ae Shin

http://www.w3.org/2001/XMLSchema#string

Myung-Jo You

http://www.w3.org/2001/XMLSchema#string

Sung-Dae Cho

http://www.w3.org/2001/XMLSchema#string

P2860

Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1

Movement of Bax from the cytosol to mitochondria during apoptosis

An overview of the rare parotid gland cancer

Stabilization and enhancement of the antiapoptotic activity of mcl-1 by TCTP

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

Distant metastasis of parotid gland tumors.

CDDO-Me, a synthetic triterpenoid, inhibits expression of IL-6 and Stat3 phosphorylation in multi-drug resistant ovarian cancer cells.

Cytotoxicity of digitoxin and related cardiac glycosides in human tumor cells.

Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.

Translational control of cell fate: availability of phosphorylation sites on translational repressor 4E-BP1 governs its proapoptotic potency.

P304

E761-70

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/HED.24096

http://www.w3.org/2001/XMLSchema#string

P478

38 Suppl 1

http://www.w3.org/2001/XMLSchema#string

P577

2015-04-27T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

25914292

http://www.w3.org/2001/XMLSchema#string