Differences in both inositol 1,4,5-trisphosphate mass and inositol 1,4,5-trisphosphate receptors between normal and dystrophic skeletal muscle cell lines.
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Dystrophic Cardiomyopathy-Potential Role of Calcium in Pathogenesis, Treatment and Novel TherapiesNifedipine treatment reduces resting calcium concentration, oxidative and apoptotic gene expression, and improves muscle function in dystrophic mdx mice.Increased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubesCell lines as in vitro models for drug screening and toxicity studies.Mini-dystrophin expression down-regulates overactivation of G protein-mediated IP3 signaling pathway in dystrophin-deficient muscle cells.Mini-dystrophin expression down-regulates IP3-mediated calcium release events in resting dystrophin-deficient muscle cells.Dihydropyridine receptors as voltage sensors for a depolarization-evoked, IP3R-mediated, slow calcium signal in skeletal muscle cellsLipid mapping in human dystrophic muscle by cluster-time-of-flight secondary ion mass spectrometry imaging.Slow calcium signals after tetanic electrical stimulation in skeletal myotubes.Copper neurotoxicity is dependent on dopamine-mediated copper uptake and one-electron reduction of aminochrome in a rat substantia nigra neuronal cell line.Optogenetic approach for targeted activation of global calcium transients in differentiated C2C12 myotubes.Bcl-2 overexpression prevents calcium overload and subsequent apoptosis in dystrophic myotubes.Involvement of TRPC in the abnormal calcium influx observed in dystrophic (mdx) mouse skeletal muscle fibers.Pacific ciguatoxin-1b effect over Na+ and K+ currents, inositol 1,4,5-triphosphate content and intracellular Ca2+ signals in cultured rat myotubes.Insulin-like growth factor-1 induces an inositol 1,4,5-trisphosphate-dependent increase in nuclear and cytosolic calcium in cultured rat cardiac myocytes.Age-dependent changes in diastolic Ca(2+) and Na(+) concentrations in dystrophic cardiomyopathy: Role of Ca(2+) entry and IP3.Negative modulation of inositol 1,4,5-trisphosphate type 1 receptor expression prevents dystrophin-deficient muscle cells death.
P2860
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P2860
Differences in both inositol 1,4,5-trisphosphate mass and inositol 1,4,5-trisphosphate receptors between normal and dystrophic skeletal muscle cell lines.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Differences in both inositol 1 ...... ic skeletal muscle cell lines.
@en
type
label
Differences in both inositol 1 ...... ic skeletal muscle cell lines.
@en
prefLabel
Differences in both inositol 1 ...... ic skeletal muscle cell lines.
@en
P2093
P2860
P1433
P1476
Differences in both inositol 1 ...... ic skeletal muscle cell lines.
@en
P2093
Caviedes R
Jaimovich E
Liberona JL
Petherbridge L
P2860
P304
P356
10.1002/(SICI)1097-4598(199807)21:7<902::AID-MUS8>3.0.CO;2-A
P577
1998-07-01T00:00:00Z