Bcl-xL acts downstream of caspase-8 activation by the CD95 death-inducing signaling complex. - Wikidata - wikimedia - TriplyDB

Bcl-xL acts downstream of caspase-8 activation by the CD95 death-inducing signaling complex.

Bcl-xL acts downstream of caspase-8 activation by the CD95 death-inducing signaling complex.

http://www.wikidata.org/entity/Q41062034

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The CED-4-homologous protein FLASH is involved in Fas-mediated activation of caspase-8 during apoptosis BAR: An apoptosis regulator at the intersection of caspases and Bcl-2 family proteins Identification of the cytolinker plectin as a major early in vivo substrate for caspase 8 during CD95- and tumor necrosis factor receptor-mediated apoptosis Glutathione peroxidase-1 protects from CD95-induced apoptosis DEDD, a novel death effector domain-containing protein, targeted to the nucleolus Mitochondrial release of caspase-2 and -9 during the apoptotic process A1 functions at the mitochondria to delay endothelial apoptosis in response to tumor necrosis factor Inactivation of caspase-8 on mitochondria of Bcl-xL-expressing MCF7-Fas cells: role for the bifunctional apoptosis regulator protein Apoptosis and the airway epithelium Two CD95 (APO-1/Fas) signaling pathways Type I and type II reactions in TRAIL-induced apoptosis -- results from dose-response studies.Platelets prevent acute hepatitis induced by anti-fas antibody.The regulation of apoptotic cell death.Caspase activation and neuroprotection in caspase-3- deficient mice after in vivo cerebral ischemia and in vitro oxygen glucose deprivation.CD95 is part of a let-7/p53/miR-34 regulatory network Fas receptor-mediated apoptosis: a clinical application?Enhancing DNA vaccine potency by coadministration of DNA encoding antiapoptotic proteins.The transcription factor Wilms tumor 1 confers resistance in myeloid leukemia cells against the proapoptotic therapeutic agent TRAIL (tumor necrosis factor α-related apoptosis-inducing ligand) by regulating the antiapoptotic protein Bcl-xL Bad can act as a key regulator of T cell apoptosis and T cell development.Protein kinase B regulates T lymphocyte survival, nuclear factor kappaB activation, and Bcl-X(L) levels in vivo Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein.Antigen activation and impaired Fas-induced death-inducing signaling complex formation in T-large-granular lymphocyte leukemia Hydroxycamptothecin induces apoptosis and inhibits tumor growth in colon cancer by the downregulation of survivin and XIAP expression Mitochondrial mechanisms of sepsis-induced organ failure.The HECTD3 E3 ubiquitin ligase facilitates cancer cell survival by promoting K63-linked polyubiquitination of caspase-8 Enhanced cell growth inhibition by thiacremonone in paclitaxel-treated lung cancer cells.The ribonucleotide reductase R1 subunits of herpes simplex virus types 1 and 2 protect cells against TNFα- and FasL-induced apoptosis by interacting with caspase-8.Molecular ordering of the initial signaling events of CD95.Proteasome inhibition results in TRAIL sensitization of primary keratinocytes by removing the resistance-mediating block of effector caspase maturation Inhibition of BCL11B expression leads to apoptosis of malignant but not normal mature T cells.Enhancement of suicidal DNA vaccine potency by delaying suicidal DNA-induced cell death.Fas, ceramide and serum withdrawal induce apoptosis via a common pathway in a type II Jurkat cell line.Caspases induce cytochrome c release from mitochondria by activating cytosolic factors.Ginsenoside Rb1 Prevents MPP(+)-Induced Apoptosis in PC12 Cells by Stimulating Estrogen Receptors with Consequent Activation of ERK1/2, Akt and Inhibition of SAPK/JNK, p38 MAPK.Up-regulation of FLIP in cisplatin-selected HeLa cells causes cross-resistance to CD95/Fas death signalling.Arsenic trioxide and breast cancer: analysis of the apoptotic, differentiative and immunomodulatory effects.Wild-type p53 induced sensitization of mutant p53 TNF-resistant cells: role of caspase-8 and mitochondria.Caspases disrupt mitochondrial membrane barrier function.Activation of Mitochondria and Release of Mitochondrial Apoptogenic Factors by Betulinic Acid

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P2860

Bcl-xL acts downstream of caspase-8 activation by the CD95 death-inducing signaling complex.

http://www.wikidata.org/entity/Q41062034

description

1998 nî lūn-bûn

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1998年の論文

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1998年学术文章

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1998年学术文章

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1998年学术文章

@zh-hans

1998年学术文章

@zh-my

1998年学术文章

@zh-sg

1998年學術文章

@yue

1998年學術文章

@zh

1998年學術文章

@zh-hant

name

Bcl-xL acts downstream of casp ...... th-inducing signaling complex.

@en

type

label

Bcl-xL acts downstream of casp ...... th-inducing signaling complex.

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prefLabel

Bcl-xL acts downstream of casp ...... th-inducing signaling complex.

@en

P1433

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P1476

Bcl-xL acts downstream of casp ...... ath-inducing signaling complex

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P2093

C Scaffidi

http://www.w3.org/2001/XMLSchema#string

J P Medema

http://www.w3.org/2001/XMLSchema#string

P H Krammer

http://www.w3.org/2001/XMLSchema#string

P2860

A novel protein that interacts with the death domain of Fas/APO1 contains a sequence motif related to the death domain

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

In vitro activation of CPP32 and Mch3 by Mch4, a novel human apoptotic cysteine protease containing two FADD-like domains

Interaction of CED-4 with CED-3 and CED-9: a molecular framework for cell death

Interaction and regulation of subcellular localization of CED-4 by CED-9

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

FLICE is activated by association with the CD95 death-inducing signaling complex (DISC).

The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1 beta-converting enzyme

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

P304

3388-3393

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scholarly article

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10.1074/JBC.273.6.3388

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6

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Marcus E. Peter

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247483485

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9452459

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