Nebivolol Desensitizes Myofilaments of a Hypertrophic Cardiomyopathy Mouse Model.

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Nebivolol Desensitizes Myofilaments of a Hypertrophic Cardiomyopathy Mouse Model.

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2017 nî lūn-bûn

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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Nebivolol Desensitizes Myofilaments of a Hypertrophic Cardiomyopathy Mouse Model.

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Nebivolol Desensitizes Myofilaments of a Hypertrophic Cardiomyopathy Mouse Model.

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Nebivolol Desensitizes Myofilaments of a Hypertrophic Cardiomyopathy Mouse Model.

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P1476

Nebivolol Desensitizes Myofilaments of a Hypertrophic Cardiomyopathy Mouse Model.

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P2093

Felix W Friedrich

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Lucie Carrier

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Nico Kresin

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Sabrina Stücker

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P2860

Advances in medical treatment of hypertrophic cardiomyopathy

Pharmacological treatment options for hypertrophic cardiomyopathy: high time for evidence

Understanding cardiomyopathy phenotypes based on the functional impact of mutations in the myosin motor

Targets for therapy in sarcomeric cardiomyopathies

Solution Structure of Human Cardiac Troponin C in Complex with the Green Tea Polyphenol, (-)-Epigallocatechin 3-Gallate

Hypertrophic cardiomyopathy: distribution of disease genes, spectrum of mutations, and implications for a molecular diagnosis strategy

Cardiac myosin-binding protein C mutations and hypertrophic cardiomyopathy: haploinsufficiency, deranged phosphorylation, and cardiomyocyte dysfunction

Cardiac myosin-binding protein C is required for complete relaxation in intact myocytes

Nonsense-mediated mRNA decay and ubiquitin-proteasome system regulate cardiac myosin-binding protein C mutant levels in cardiomyopathic mice

Mybpc3 gene therapy for neonatal cardiomyopathy enables long-term disease prevention in mice

P304

558

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scholarly article

P356

10.3389/FPHYS.2017.00558

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P478

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P577

2017-08-02T00:00:00Z

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P698

28824454

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P932

5539082

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