Extracellular glycine is necessary for optimal hemoglobinization of erythroid cells.

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Glutamine via α-ketoglutarate dehydrogenase provides succinyl-CoA for heme synthesis during erythropoiesis

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Extracellular glycine is necessary for optimal hemoglobinization of erythroid cells.

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Extracellular glycine is necessary for optimal hemoglobinization of erythroid cells.

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Extracellular glycine is necessary for optimal hemoglobinization of erythroid cells.

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Extracellular glycine is necessary for optimal hemoglobinization of erythroid cells.

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P1476

Extracellular glycine is necessary for optimal hemoglobinization of erythroid cells.

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Alex D Sheftel

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Daniel Garcia-Santos

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Matthias Schranzhofer

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Prem Ponka

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Mutations in mitochondrial carrier family gene SLC25A38 cause nonsyndromic autosomal recessive congenital sideroblastic anemia

Ontogeny of erythroid gene expression

Glycine and Folate Ameliorate Models of Congenital Sideroblastic Anemia

The monoclonal antibody TER-119 recognizes a molecule associated with glycophorin A and specifically marks the late stages of murine erythroid lineage

Catabolism of threonine in mammals by coupling of L-threonine 3-dehydrogenase with 2-amino-3-oxobutyrate-CoA ligase

Beyond the dopamine receptor: novel therapeutic targets for treating schizophrenia.

Gene knockout of glycine transporter 1: characterization of the behavioral phenotype.

Glycine supply to human enterocytes mediated by high-affinity basolateral GLYT1.

A weak link in metabolism: the metabolic capacity for glycine biosynthesis does not satisfy the need for collagen synthesis.

Glycine transporters as novel therapeutic targets in schizophrenia, alcohol dependence and pain.

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scholarly article

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10.3324/HAEMATOL.2016.155671

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102

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R Bergeron

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2017-05-11T00:00:00Z

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28495915

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5541866

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Extracellular glycine is necessary for optimal hemoglobinization of erythroid cells.

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P2860

P2860

Extracellular glycine is necessary for optimal hemoglobinization of erythroid cells.

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