Association of the invariant chain with major histocompatibility complex class I molecules directs trafficking to endocytic compartments.
about
Direct binding of human immunodeficiency virus type 1 Nef to the major histocompatibility complex class I (MHC-I) cytoplasmic tail disrupts MHC-I traffickingMHC class I endosomal and lysosomal trafficking coincides with exogenous antigen loading in dendritic cellsImmature dendritic cells phagocytose apoptotic cells via alphavbeta5 and CD36, and cross-present antigens to cytotoxic T lymphocytesPathways of antigen processing.Cytomegaloviral control of MHC class I function in the mouse.An in vitro study of the dynamic features of the major histocompatibility complex class I complex relevant to its role as a versatile peptide-receptive moleculeIntracellular traffic to compartments for MHC class II peptide loading: signals for endosomal and polarized sorting.Promiscuous binding of invariant chain-derived CLIP peptide to distinct HLA-I molecules revealed in leukemic cells.Cross-presentation: underlying mechanisms and role in immune surveillance.Transporters associated with antigen processing (TAP)-independent presentation of soluble insulin to alpha/beta T cells by the class Ib gene product, Qa-1(b).Presentation of peptide antigens by mouse CD1 requires endosomal localization and protein antigen processing.A CD74-dependent MHC class I endolysosomal cross-presentation pathway.The MHC class II-associated invariant chain interacts with the neonatal Fc gamma receptor and modulates its trafficking to endosomal/lysosomal compartments.CD1c molecules broadly survey the endocytic systemEffect of invariant chain on major histocompatibility complex class I molecule expression and stability on human breast tumor cell lines.Physical and functional interaction of transmembrane thioredoxin-related protein with major histocompatibility complex class I heavy chain: redox-based protein quality control and its potential relevance to immune responsesBiosynthesis of major histocompatibility complex molecules and generation of T cells in Ii TAP1 double-mutant mice.TAPBPR: a new player in the MHC class I presentation pathway.The comings and goings of MHC class I molecules herald a new dawn in cross-presentation.The Biology and Underlying Mechanisms of Cross-Presentation of Exogenous Antigens on MHC-I Molecules.The endogenous pathway of MHC class II antigen presentation.Endosomal compartment: Also a dock for MHC class I peptide loading.A cytomegalovirus glycoprotein re-routes MHC class I complexes to lysosomes for degradation.Carbon monoxide decreases endosome-lysosome fusion and inhibits soluble antigen presentation by dendritic cells to T cells.Cross presentation of antigen by dendritic cells: mechanisms and implications for immunotherapy.Invariant chain as a vehicle to load antigenic peptides on human MHC class I for cytotoxic T-cell activation.Development of a Molecular Adjuvant to Enhance Antigen-Specific CD8 T Cell Responses
P2860
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P2860
Association of the invariant chain with major histocompatibility complex class I molecules directs trafficking to endocytic compartments.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Association of the invariant c ...... ing to endocytic compartments.
@en
type
label
Association of the invariant c ...... ing to endocytic compartments.
@en
prefLabel
Association of the invariant c ...... ing to endocytic compartments.
@en
P2860
P356
P1476
Association of the invariant c ...... ing to endocytic compartments.
@en
P2093
P2860
P304
P356
10.1074/JBC.270.3.1443
P407
P577
1995-01-01T00:00:00Z