Neuronal Atrophy Early in Degenerative Ataxia Is a Compensatory Mechanism to Regulate Membrane Excitability.
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Precision medicine in spinocerebellar ataxias: treatment based on common mechanisms of diseaseEmerging Roles of Filopodia and Dendritic Spines in Motoneuron Plasticity during Development and DiseaseBK Channels in the Central Nervous System.Heat Shock Protein Beta-1 Modifies Anterior to Posterior Purkinje Cell Vulnerability in a Mouse Model of Niemann-Pick Type C DiseaseIn vivo analysis of cerebellar Purkinje cell activity in SCA2 transgenic mouse model.4-aminopyridine reverses ataxia and cerebellar firing deficiency in a mouse model of spinocerebellar ataxia type 6.Degenerative Ataxias: challenges in clinical research.Posterior cerebellar Purkinje cells in an SCA5/SPARCA1 mouse model are especially vulnerable to the synergistic effect of loss of β-III spectrin and GLAST.Cellular and circuit mechanisms underlying spinocerebellar ataxias.Progressive impairment of cerebellar mGluR signalling and its therapeutic potential for cerebellar ataxia in spinocerebellar ataxia type 1 model mice.Molecular Determinants of BK Channel Functional Diversity and Functioning.Brain atrophy in picornavirus-infected FVB mice is dependent on the H-2Db class I molecule.Polyglutamine spinocerebellar ataxias - from genes to potential treatments.Expression of mutant DISC1 in Purkinje cells increases their spontaneous activity and impairs cognitive and social behaviors in mice.New old drug(s) for spinocerebellar ataxias.Transient cerebellar alterations during development prior to obvious motor phenotype in a mouse model of spinocerebellar ataxia type 6.Ion channel dysfunction in cerebellar ataxia.Potassium channel dysfunction underlies Purkinje neuron spiking abnormalities in spinocerebellar ataxia type 2.Neurodegenerative disease: downsizing neurons.Targeting potassium channels to treat cerebellar ataxia.Genetic Ablation of EWS RNA Binding Protein 1 (EWSR1) Leads to Neuroanatomical Changes and Motor Dysfunction in Mice.Dendritic potassium channel dysfunction may contribute to dendrite degeneration in spinocerebellar ataxia type 1.The Emerging Concept of Intrinsic Plasticity: Activity-dependent Modulation of Intrinsic Excitability in Cerebellar Purkinje Cells and Motor Learning.Altered synaptic and firing properties of cerebellar Purkinje cells in a mouse model of ARSACS
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P2860
Neuronal Atrophy Early in Degenerative Ataxia Is a Compensatory Mechanism to Regulate Membrane Excitability.
description
2015 nî lūn-bûn
@nan
2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
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2015年论文
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name
Neuronal Atrophy Early in Dege ...... egulate Membrane Excitability.
@en
type
label
Neuronal Atrophy Early in Dege ...... egulate Membrane Excitability.
@en
prefLabel
Neuronal Atrophy Early in Dege ...... egulate Membrane Excitability.
@en
P2093
P2860
P50
P1476
Neuronal Atrophy Early in Dege ...... Regulate Membrane Excitability
@en
P2093
Aaron H Wasserman
Heike Wulff
Melissa A C Ingram
Puneet Opal
Ravi Chopra
Vikram G Shakkottai
Vikrant Singh
P2860
P304
11292-11307
P356
10.1523/JNEUROSCI.1357-15.2015
P407
P577
2015-08-01T00:00:00Z