Studies of hypercholesterolemia in the nonhuman primate. II. Fatty streak conversion to fibrous plaque.
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Coexpression of type I and type II human macrophage scavenger receptors in macrophages of various organs and foam cells in atherosclerotic lesionsMolecular Imaging of Vulnerable Atherosclerotic Plaques in Animal Models.Efficient lytic infection of human arterial endothelial cells by human cytomegalovirus strainsIn vivo interrogation of the molecular display of atherosclerotic lesion surfaces.A role for endothelial cell lipoxygenase in the oxidative modification of low density lipoproteinMorphology of the endothelium over atherosclerotic plaques in human coronary arteries.Inhibition of hypercholesterolemia-induced atherosclerosis in the nonhuman primate by probucol. I. Is the extent of atherosclerosis related to resistance of LDL to oxidation?Interleukin 1 acts on cultured human vascular endothelium to increase the adhesion of polymorphonuclear leukocytes, monocytes, and related leukocyte cell linesMetabolism of very low density lipoproteins after cessation of cholesterol feeding in rabbits. A factor potentially contributing to the slow regression of atheromatous plaques.New mechanism for foam cell generation in atherosclerotic lesions.Role of platelets in cholesteryl ester formation by U-937 cellsStimulation of aortic smooth muscle cell mitogenesis by serotonin.Formation of factor Va by atherosclerotic rabbit aorta mediates factor Xa-catalyzed prothrombin activationA practical approach to using mice in atherosclerosis research.Blockade of platelet-derived growth factor or its receptors transiently delays but does not prevent fibrous cap formation in ApoE null mice.Interleukin 1: a mitogen for human vascular smooth muscle cells that induces the release of growth-inhibitory prostanoids.Macrophage secretory products selectively stimulate dermatan sulfate proteoglycan production in cultured arterial smooth muscle cellsDerivation and properties of platelet-derived growth factor-independent rat smooth muscle cells.Human atherosclerosis. III. Immunocytochemical analysis of the cell composition of lesions of young adultsInduction of fatty streak-like lesions in vitro using a culture model system simulating arterial intima.Rous-Whipple Award Lecture. Atherosclerosis: a defense mechanism gone awry.Transitional features in human atherosclerosis. Intimal thickening, cholesterol clefts, and cell loss in human aortic fatty streaksEarly atherogenesis in White Carneau pigeons. II. Ultrastructural and cytochemical observationsMorphology and cell kinetics of fatty streak lesion formation in the hypercholesterolemic rabbit.Activation of coagulation releases endothelial cell mitogensHuman interleukin 1 induces interleukin 1 gene expression in human vascular smooth muscle cells.Protocatechuic aldehyde inhibits TNF-α-induced fibronectin expression in human umbilical vein endothelial cells via a c-Jun N-terminal kinase dependent pathwayCoagulation factors X, Xa, and protein S as potent mitogens of cultured aortic smooth muscle cells.Aortic endothelial cells regulate proliferation of human monocytes in vitro via a mechanism synergistic with macrophage colony-stimulating factor. Convergence at the cyclin E/p27(Kip1) regulatory checkpointFatty streak formation occurs in human fetal aortas and is greatly enhanced by maternal hypercholesterolemia. Intimal accumulation of low density lipoprotein and its oxidation precede monocyte recruitment into early atherosclerotic lesions.Macrophages and smooth muscle cells express lipoprotein lipase in human and rabbit atherosclerotic lesions.Hypolipidemic activity of a natural mineral water rich in calcium, magnesium, and bicarbonate in hyperlipidemic adults.Antioxidant treatment inhibits the development of intimal thickening after balloon injury of the aorta in hypercholesterolemic rabbits.Mechanisms of monocyte recruitment and accumulation.Atherosclerosis: what is it and why does it occur?Two distinct receptors account for recognition of maleyl-albumin in human monocytes during differentiation in vitro.Interactions of monocytic cells with human endothelial cells stimulate monocytic metalloproteinase production.Decreased blood flow rate disrupts endothelial repair in vivo.Risk of thrombosis in human atherosclerotic plaques: role of extracellular lipid, macrophage, and smooth muscle cell content.Atherosclerosis, platelets and thrombosis in acute ischaemic heart disease.
P2860
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P2860
Studies of hypercholesterolemia in the nonhuman primate. II. Fatty streak conversion to fibrous plaque.
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
Studies of hypercholesterolemi ...... conversion to fibrous plaque.
@en
type
label
Studies of hypercholesterolemi ...... conversion to fibrous plaque.
@en
prefLabel
Studies of hypercholesterolemi ...... conversion to fibrous plaque.
@en
P356
P1476
Studies of hypercholesterolemi ...... k conversion to fibrous plaque
@en
P2093
P304
P356
10.1161/01.ATV.4.4.341
P577
1984-07-01T00:00:00Z