A post-transcriptional compensatory pathway in heterozygous ventricular myosin light chain 2-deficient mice results in lack of gene dosage effect during normal cardiac growth or hypertrophy.
about
Phosphorylation of the regulatory light chain of myosin in striated muscle: methodological perspectivesHypoxia-inducible factor-dependent degeneration, failure, and malignant transformation of the heart in the absence of the von Hippel-Lindau proteinThe in vivo role of p38 MAP kinases in cardiac remodeling and restrictive cardiomyopathyPak1 as a novel therapeutic target for antihypertrophic treatment in the heartConditional mutation of the ErbB2 (HER2) receptor in cardiomyocytes leads to dilated cardiomyopathyAllele interaction--single locus genetics meets regulatory biologyAtrial chamber-specific expression of sarcolipin is regulated during development and hypertrophic remodeling.Impaired Ca2+ store functions in skeletal and cardiac muscle cells from sarcalumenin-deficient mice.Cardiomyocyte-specific prolyl-4-hydroxylase domain 2 knock out protects from acute myocardial ischemic injuryMice carrying a conditional Serca2(flox) allele for the generation of Ca(2+) handling-deficient mouse models.Derivation of functional ventricular cardiomyocytes using endogenous promoter sequence from murine embryonic stem cells.A cardiac myocyte vascular endothelial growth factor paracrine pathway is required to maintain cardiac function.Central role of mitofusin 2 in autophagosome-lysosome fusion in cardiomyocytes.Crim1 has cell-autonomous and paracrine roles during embryonic heart development.Abnormalities of motor function, transcription and cerebellar structure in mouse models of THAP1 dystonia.Role of VEGF in organogenesis.B-myb is required for inner cell mass formation at an early stage of development.Post-transcriptional downregulation of sarcolipin mRNA by triiodothyronine in the atrial myocardium.Direct inhibition of type 5 adenylyl cyclase prevents myocardial apoptosis without functional deterioration.Desmosomal junctions are necessary for adult sinus node function.Heterozygous deletion of sarcolipin maintains normal cardiac function.Dilated cardiomyopathy in Erb-b4-deficient ventricular muscle.Targeted activation of c-Jun N-terminal kinase in vivo induces restrictive cardiomyopathy and conduction defects.Endoglin haploinsufficiency is associated with differential regulation of extracellular matrix production during skin fibrosis and cartilage repair in mice.
P2860
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P2860
A post-transcriptional compensatory pathway in heterozygous ventricular myosin light chain 2-deficient mice results in lack of gene dosage effect during normal cardiac growth or hypertrophy.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
A post-transcriptional compens ...... cardiac growth or hypertrophy.
@en
type
label
A post-transcriptional compens ...... cardiac growth or hypertrophy.
@en
prefLabel
A post-transcriptional compens ...... cardiac growth or hypertrophy.
@en
P2093
P356
P1476
A post-transcriptional compens ...... cardiac growth or hypertrophy
@en
P2093
P304
10066-10070
P356
10.1074/JBC.274.15.10066
P407
P577
1999-04-01T00:00:00Z