Depletion of c-myc with specific antisense sequences reverses the transformed phenotype in ras oncogene-transformed NIH 3T3 cells.
about
BIN1 is a novel MYC-interacting protein with features of a tumour suppressorActivation of extracellular signal-regulated kinase, ERK2, by p21ras oncoproteinLoss of transgelin in breast and colon tumors and in RIE-1 cells by Ras deregulation of gene expression through Raf-independent pathwaysActivation of cyclin-dependent kinases by Myc mediates induction of cyclin A, but not apoptosisRaman spectroscopic study of radioresistant oral cancer sublines established by fractionated ionizing radiation.New Myc-interacting proteins: a second Myc network emerges.Mechanisms of apoptosis by c-Myc.CCAAT/enhancer binding protein-beta is a mediator of keratinocyte survival and skin tumorigenesis involving oncogenic Ras signalingConcepts and strategies for human gene therapy.Adenoviral vectors: development and application.Strategies to Inhibit Myc and Their Clinical ApplicabilityAn essential E box in the promoter of the gene encoding the mRNA cap-binding protein (eukaryotic initiation factor 4E) is a target for activation by c-myc.Regulation of collagen I gene expression by rasCell cycle-dependent phosphorylation of C/EBPbeta mediates oncogenic cooperativity between C/EBPbeta and H-RasV12.Complementation of defective colony-stimulating factor 1 receptor signaling and mitogenesis by Raf and v-Src.Gene therapy in surgical oncology.Regulation of the junB gene by v-src.Promotion of growth and apoptosis in c-myc nullizygous fibroblasts by other members of the myc oncoprotein family.Effects of an inducible anti-sense c-myc gene transfer in a drug-resistant human small-cell-lung-carcinoma cell line.A human T lymphotropic virus type I (HTLV-I) long terminal repeat-directed antisense c-myc construct with an Epstein-Barr virus replicon vector inhibits cell growth in a HTLV-I-transformed human T cell line.Mmip-2/Rnf-17 enhances c-Myc function and regulates some target genes in common with glucocorticoid hormones.Tissue-selective expression of dominant-negative proteins for the regulation of vascular smooth muscle cell proliferation.Constitutive activation and overexpression of NF-κB/c-Rel in conjunction with p50 contribute to aggressive tongue tumorigenesis
P2860
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P2860
Depletion of c-myc with specific antisense sequences reverses the transformed phenotype in ras oncogene-transformed NIH 3T3 cells.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Depletion of c-myc with specif ...... ene-transformed NIH 3T3 cells.
@en
type
label
Depletion of c-myc with specif ...... ene-transformed NIH 3T3 cells.
@en
prefLabel
Depletion of c-myc with specif ...... ene-transformed NIH 3T3 cells.
@en
P2093
P2860
P356
P1476
Depletion of c-myc with specif ...... ene-transformed NIH 3T3 cells.
@en
P2093
E Thompson
E V Prochownik
H B Grossman
P2860
P304
P356
10.1128/MCB.11.7.3699
P407
P577
1991-07-01T00:00:00Z