Training-induced elevation in FABP(PM) is associated with increased palmitate use in contracting muscle.
about
Intramuscular triacylglycerol, glycogen and acetyl group metabolism during 4 h of moderate exercise in manFasting for 72 h increases intramyocellular lipid content in nondiabetic, physically fit men.Two weeks of moderate-intensity continuous training, but not high-intensity interval training, increases insulin-stimulated intestinal glucose uptakeMetabolic syndrome and insulin resistance: underlying causes and modification by exercise training.Is membrane transport of FFA mediated by lipid, protein, or both? Mechanisms and regulation of protein-mediated cellular fatty acid uptake: molecular, biochemical, and physiological evidence.Skeletal muscle insulin resistance: roles of fatty acid metabolism and exerciseShort-term AMP-regulated protein kinase activation enhances insulin-sensitive fatty acid uptake and increases the effects of insulin on fatty acid oxidation in L6 muscle cells.Oxidation of Intracellular and Extracellular Fatty Acids in Skeletal Muscle: Application of kinetic modeling, stable isotopes and liquid chromatography/electrospray ionization ion-trap tandem mass spectrometry technology.CaMKK is an upstream signal of AMP-activated protein kinase in regulation of substrate metabolism in contracting skeletal muscle.High carbohydrate availability increases LCFA uptake and decreases LCFA oxidation in perfused muscle.Brief food restriction increases FA oxidation and glycogen synthesis under insulin-stimulated conditions.Impaired fatty acid oxidation in muscle of aging rats perfused under basal conditions.Insulin fails to alter plasma LCFA metabolism in muscle perfused at similar glucose uptake.AMPKα2 deficiency uncovers time dependency in the regulation of contraction-induced palmitate and glucose uptake in mouse muscle.Aging is associated with elevated muscle triglyceride content and increased insulin-stimulated fatty acid uptake.AMPK activation is not critical in the regulation of muscle FA uptake and oxidation during low-intensity muscle contraction.AMPK-α2 is involved in exercise training-induced adaptations in insulin-stimulated metabolism in skeletal muscle following high-fat diet.Thiazolidinediones enhance skeletal muscle triacylglycerol synthesis while protecting against fatty acid-induced inflammation and insulin resistance.Insulin-stimulated plasma membrane association and activation of Akt2, aPKC zeta and aPKC lambda in high fat fed rodent skeletal muscle.
P2860
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P2860
Training-induced elevation in FABP(PM) is associated with increased palmitate use in contracting muscle.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Training-induced elevation in ...... ate use in contracting muscle.
@en
type
label
Training-induced elevation in ...... ate use in contracting muscle.
@en
prefLabel
Training-induced elevation in ...... ate use in contracting muscle.
@en
P2093
P2860
P1476
Training-induced elevation in ...... ate use in contracting muscle.
@en
P2093
J R Swenberger
L P Turcotte
M Z Tucker
P2860
P304
P356
10.1152/JAPPL.1999.87.1.285
P407
P577
1999-07-01T00:00:00Z