Antigen presentation requires transport of MHC class I molecules from the endoplasmic reticulum.
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Processing of urushiol (poison ivy) hapten by both endogenous and exogenous pathways for presentation to T cells in vitroIdentification of human cancers deficient in antigen processingRevisiting the Middle Molecule Hypothesis of Uremic Toxicity: A Systematic Review of Beta 2 Microglobulin Population Kinetics and Large Scale Modeling of Hemodialysis Trials In SilicoViral inhibition of the transporter associated with antigen processing (TAP): a striking example of functional convergent evolutionHierarchy among multiple H-2b-restricted cytotoxic T-lymphocyte epitopes within simian virus 40 T antigen.Human cytomegalovirus UL18 utilizes US6 for evading the NK and T-cell responsesSalmonella typhimurium delta aroA delta aroD mutants expressing a foreign recombinant protein induce specific major histocompatibility complex class I-restricted cytotoxic T lymphocytes in mice.Genetically engineered pancreatic beta-cell lines for cell therapy of diabetes.Rediscovering Beta-2 Microglobulin As a Biomarker across the Spectrum of Kidney Diseases.Prolonged survival of pancreatic islet allografts mediated by adenovirus immunoregulatory transgenes.The endoplasmic reticulum lumenal domain of the adenovirus type 2 E3-19K protein binds to peptide-filled and peptide-deficient HLA-A*1101 molecules.The MHC class I antigen presentation pathway: strategies for viral immune evasion.The early expression of glycoprotein B from herpes simplex virus can be detected by antigen-specific CD8+ T cells.Insights into the paracrine effects of uterine natural killer cells.Determinants of the endoplasmic reticulum (ER) lumenal-domain of the adenovirus serotype 2 E3-19K protein for association with and ER-retention of major histocompatibility complex class I moleculesAdenovirus E3-19K proteins of different serotypes and subgroups have similar, yet distinct, immunomodulatory functions toward major histocompatibility class I moleculesFunctions and mechanisms of action of the adenovirus E3 proteins.Region E3 of subgroup B human adenoviruses encodes a 16-kilodalton membrane protein that may be a distant analog of the E3-6.7K protein of subgroup C adenoviruses.Early region 3 of adenovirus type 19 (subgroup D) encodes an HLA-binding protein distinct from that of subgroups B and CExpression of gp19K increases the persistence of transgene expression from an adenovirus vector in the mouse lung and liver.Defective presentation of endogenous antigens by a murine sarcoma. Implications for the failure of an anti-tumor immune responseRecognition of vaccinia virus-encoded major histocompatibility complex class I antigens by virus immune cytotoxic T cells is independent of the polymorphism of the peptide transporters.TAP-1 indirectly regulates CD4+ T cell priming in Toxoplasma gondii infection by controlling NK cell IFN-gamma production.Retention of adenovirus E19 glycoprotein in the endoplasmic reticulum is essential to its ability to block antigen presentation.Flanking sequences influence the presentation of an endogenously synthesized peptide to cytotoxic T lymphocytes.Cytotoxic T cell recognition of an endogenous class I HLA peptide presented by a class II HLA moleculeAn endogenous processing pathway in vaccinia virus-infected cells for presentation of cytoplasmic antigens to class II-restricted T cellsClass I molecules retained in the endoplasmic reticulum bind antigenic peptidesComparison of cell lines deficient in antigen presentation reveals a functional role for TAP-1 alone in antigen processing.Heat-shock protein 90 associates with N-terminal extended peptides and is required for direct and indirect antigen presentationThe isolated ER-Golgi intermediate compartment exhibits properties that are different from ER and cis-Golgi.Hepatitis B surface antigen assembles in a post-ER, pre-Golgi compartment.HLA class I heavy-chain gene promoter elements mediating synergy between tumor necrosis factor and interferons.Deletion mutation analysis of the adenovirus type 2 E3-gp19K protein: identification of sequences within the endoplasmic reticulum lumenal domain that are required for class I antigen binding and protection from adenovirus-specific cytotoxic T lymphSimian virus 40 T antigen as a carrier for the expression of cytotoxic T-lymphocyte recognition epitopes.Vascular endothelial growth factor C facilitates immune tolerance and endovascular activity of human uterine NK cells at the maternal-fetal interfaceConformational constraints involved in MHC class I restricted antigen presentation.Cross-presenting human gammadelta T cells induce robust CD8+ alphabeta T cell responses.Induction of antigen-specific class I-restricted cytotoxic T cells by soluble proteins in vivo.Tumor necrosis factor alpha stimulates expression of adenovirus early region 3 proteins: implications for viral persistence.
P2860
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P2860
Antigen presentation requires transport of MHC class I molecules from the endoplasmic reticulum.
description
1990 nî lūn-bûn
@nan
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
1990年论文
@zh
1990年论文
@zh-cn
name
Antigen presentation requires ...... rom the endoplasmic reticulum.
@en
type
label
Antigen presentation requires ...... rom the endoplasmic reticulum.
@en
prefLabel
Antigen presentation requires ...... rom the endoplasmic reticulum.
@en
P2093
P356
P1433
P1476
Antigen presentation requires ...... rom the endoplasmic reticulum.
@en
P2093
P304
P356
10.1126/SCIENCE.2137259
P407
P577
1990-02-01T00:00:00Z