Atypical axon-Schwann cell relationships in the common peroneal nerve of the dystrophic mouse: an ultrastructural study.
about
Distinct roles for laminin globular domains in laminin alpha1 chain mediated rescue of murine laminin alpha2 chain deficiencyFactors influencing the release of proteins by cultured Schwann cells.Schwann cells proliferate but fail to differentiate in defined mediumRole of integrins in peripheral nerves and hereditary neuropathies.Biological role of dystroglycan in Schwann cell function and its implications in peripheral nervous system diseases.Laminin alpha1 chain improves laminin alpha2 chain deficient peripheral neuropathy.Normal basal laminas are realized on dystrophic Schwann cells in dystrophic in equilibrium shiverer chimera nervesBoth laminin and Schwann cell dystroglycan are necessary for proper clustering of sodium channels at nodes of Ranvier.
P2860
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P2860
Atypical axon-Schwann cell relationships in the common peroneal nerve of the dystrophic mouse: an ultrastructural study.
description
1979 nî lūn-bûn
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1979年の論文
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1979年論文
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1979年論文
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1979年論文
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1979年論文
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1979年論文
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1979年论文
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1979年论文
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1979年论文
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name
Atypical axon-Schwann cell rel ...... use: an ultrastructural study.
@en
type
label
Atypical axon-Schwann cell rel ...... use: an ultrastructural study.
@en
prefLabel
Atypical axon-Schwann cell rel ...... use: an ultrastructural study.
@en
P2860
P1476
Atypical axon-Schwann cell rel ...... use: an ultrastructural study.
@en
P2093
P2860
P304
P356
10.1111/J.1365-2990.1979.TB00666.X
P577
1979-03-01T00:00:00Z