Hypoxia reduces MAX expression in endothelial cells by unproductive splicing.
about
Hypoxia-driven splicing into noncoding isoforms regulates the DNA damage response.Design and implementation of a synthetic pre-miR switch for controlling miRNA biogenesis in mammals.Auto- and crossregulation of the hnRNPs D and DL.Riboswitching with ciprofloxacin-development and characterization of a novel RNA regulator.A small, portable RNA device for the control of exon skipping in mammalian cells.Hypoxia leads to significant changes in alternative splicing and elevated expression of CLK splice factor kinases in PC3 prostate cancer cells.
P2860
Hypoxia reduces MAX expression in endothelial cells by unproductive splicing.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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name
Hypoxia reduces MAX expression in endothelial cells by unproductive splicing.
@en
type
label
Hypoxia reduces MAX expression in endothelial cells by unproductive splicing.
@en
prefLabel
Hypoxia reduces MAX expression in endothelial cells by unproductive splicing.
@en
P2860
P1433
P1476
Hypoxia reduces MAX expression in endothelial cells by unproductive splicing
@en
P2093
Katrin Kemmerer
P2860
P304
P356
10.1016/J.FEBSLET.2014.11.011
P407
P577
2014-11-15T00:00:00Z