Dissection of the functions of the Saccharomyces cerevisiae RAD6 postreplicative repair group in mutagenesis and UV sensitivity.
about
Eukaryotic DNA polymerase ζA role for histone H2B during repair of UV-induced DNA damage in Saccharomyces cerevisiae.Roles of RAD6 epistasis group members in spontaneous polzeta-dependent translesion synthesis in Saccharomyces cerevisiaeFormation and repair of interstrand cross-links in DNADNA repair mechanisms and the bypass of DNA damage in Saccharomyces cerevisiaeRequirement of Rad5 for DNA polymerase zeta-dependent translesion synthesis in Saccharomyces cerevisiaeAnc1, a protein associated with multiple transcription complexes, is involved in postreplication repair pathway in S. cerevisiae.Shared genetic pathways contribute to the tolerance of endogenous and low-dose exogenous DNA damage in yeast.Delineating the requirements for spontaneous DNA damage resistance pathways in genome maintenance and viability in Saccharomyces cerevisiae.Yeast MPH1 gene functions in an error-free DNA damage bypass pathway that requires genes from Homologous recombination, but not from postreplicative repair.Error-free DNA damage tolerance and sister chromatid proximity during DNA replication rely on the Polα/Primase/Ctf4 ComplexPostreplication repair inhibits CAG.CTG repeat expansions in Saccharomyces cerevisiae.The mechanism of nucleotide excision repair-mediated UV-induced mutagenesis in nonproliferating cells.Mutagenic and recombinagenic responses to defective DNA polymerase delta are facilitated by the Rev1 protein in pol3-t mutants of Saccharomyces cerevisiae.The post-replication repair RAD18 and RAD6 genes are involved in the prevention of spontaneous mutations caused by 7,8-dihydro-8-oxoguanine in Saccharomyces cerevisiae.Phosphorylation of Rad55 on serines 2, 8, and 14 is required for efficient homologous recombination in the recovery of stalled replication forks.Rev1 is essential for DNA damage tolerance and non-templated immunoglobulin gene mutation in a vertebrate cell line.Non-homologous end joining as an important mutagenic process in cell cycle-arrested cellsAnalysis of Saccharomyces cerevisiae null allele strains identifies a larger role for DNA damage versus oxidative stress pathways in growth inhibition by selenium.
P2860
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P2860
Dissection of the functions of the Saccharomyces cerevisiae RAD6 postreplicative repair group in mutagenesis and UV sensitivity.
description
2001 nî lūn-bûn
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2001年の論文
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2001年論文
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2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
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2001年论文
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name
Dissection of the functions of ...... utagenesis and UV sensitivity.
@en
type
label
Dissection of the functions of ...... utagenesis and UV sensitivity.
@en
prefLabel
Dissection of the functions of ...... utagenesis and UV sensitivity.
@en
P2093
P2860
P1433
P1476
Dissection of the functions of ...... utagenesis and UV sensitivity.
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P2093
P2860
P304
P407
P577
2001-11-01T00:00:00Z