A new scorpion toxin (BmK-PL) stimulates Ca2+-release channel activity of the skeletal-muscle ryanodine receptor by an indirect mechanism.
about
Cardiac hypertrophy and impaired relaxation in transgenic mice overexpressing triadin 1U-shaped dose-dependent effects of BmK AS, a unique scorpion polypeptide toxin, on voltage-gated sodium channels.Identification of triadin 1 as the predominant triadin isoform expressed in mammalian myocardium.Hemicalcin, a new toxin from the Iranian scorpion Hemiscorpius lepturus which is active on ryanodine-sensitive Ca2+ channelsMolecular determination of selectivity of the site 3 modulator (BmK I) to sodium channels in the CNS: a clue to the importance of Nav1.6 in BmK I-induced neuronal hyperexcitability.Expression of dihydropyridine and ryanodine receptors in type IIA fibers of rat skeletal muscle.
P2860
A new scorpion toxin (BmK-PL) stimulates Ca2+-release channel activity of the skeletal-muscle ryanodine receptor by an indirect mechanism.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
A new scorpion toxin (BmK-PL) ...... ptor by an indirect mechanism.
@en
type
label
A new scorpion toxin (BmK-PL) ...... ptor by an indirect mechanism.
@en
prefLabel
A new scorpion toxin (BmK-PL) ...... ptor by an indirect mechanism.
@en
P2093
P2860
P1433
P1476
A new scorpion toxin (BmK-PL) ...... ptor by an indirect mechanism.
@en
P2093
Furukawa K
Hirayama Y
Kuniyasu A
Nakayama H
P2860
P304
P356
10.1042/0264-6021:3390343
P407
P478
339 ( Pt 2)
P577
1999-04-01T00:00:00Z