'Pore' formation is not required for the hydroperoxide-induced Ca2+ release from rat liver mitochondria. - Wikidata - wikimedia - TriplyDB

'Pore' formation is not required for the hydroperoxide-induced Ca2+ release from rat liver mitochondria.

'Pore' formation is not required for the hydroperoxide-induced Ca2+ release from rat liver mitochondria.

http://www.wikidata.org/entity/Q41841377

about

Pro-oxidants and mitochondrial Ca2+: their relationship to apoptosis and oncogenesis.The permeability transition pore as a mitochondrial calcium release channel: a critical appraisal.Nitric oxide and its congeners in mitochondria: implications for apoptosis.Cyclosporin A blocks 6-hydroxydopamine-induced efflux of Ca2+ from mitochondria without inactivating the mitochondrial inner-membrane pore Oxidative damage to mitochondria is mediated by the Ca(2+)-dependent inner-membrane permeability transition.N-acetylcysteine impairs survival of luteal cells through mitochondrial dysfunction.Effect of ebselen on Ca2+ transport in mitochondria.On the role of the respiratory complex I on membrane permeability transition.Control of the pro-oxidant-dependent calcium release from intact liver mitochondria.Peroxynitrite causes calcium efflux from mitochondria which is prevented by Cyclosporin A.Ca(2+)-induced mitochondrial membrane permeabilization: role of coenzyme Q redox state.Inhibition of the pyridine nucleotide-linked mitochondrial Ca2+release by 4-hydroxynonenal: the role of thiolate-disulfide conversion

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P2860

'Pore' formation is not required for the hydroperoxide-induced Ca2+ release from rat liver mitochondria.

http://www.wikidata.org/entity/Q41841377

description

1992 nî lūn-bûn

@nan

1992年の論文

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1992年論文

@yue

1992年論文

@zh-hant

1992年論文

@zh-hk

1992年論文

@zh-mo

1992年論文

@zh-tw

1992年论文

@wuu

1992年论文

@zh

1992年论文

@zh-cn

name

'Pore' formation is not requir ...... e from rat liver mitochondria.

@en

type

label

'Pore' formation is not requir ...... e from rat liver mitochondria.

@en

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'Pore' formation is not requir ...... e from rat liver mitochondria.

@en

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Biochemical Journal

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'Pore' formation is not requir ...... e from rat liver mitochondria.

@en

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Richter C

http://www.w3.org/2001/XMLSchema#string

Schlegel J

http://www.w3.org/2001/XMLSchema#string

P2860

Oxidative stress in mitochondria: its relationship to cellular Ca2+ homeostasis, cell death, proliferation, and differentiation.

Mechanisms by which mitochondria transport calcium.

Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

The entrapment of the Ca2+ indicator arsenazo III in the matrix space of rat liver mitochondria by permeabilization and resealing. Na+-dependent and -independent effluxes of Ca2+ in arsenazo III-loaded mitochondria.

The regulation of extramitochondrial free calcium ion concentration by rat liver mitochondria.

Ca2+ release from mitochondria induced by prooxidants.

Intracellular calcium homeostasis.

The calcium cycle of mitochondria.

A heart mitochondrial Ca2(+)-dependent pore of possible relevance to re-perfusion-induced injury. Evidence that ADP facilitates pore interconversion between the closed and open states.

Release of mitochondrial matrix proteins through a Ca2+-requiring, cyclosporin-sensitive pathway.

P304

65-69

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scholarly article

P356

10.1042/BJ2850065

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P478

285 ( Pt 1)

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Matthias Schweizer

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1992-07-01T00:00:00Z

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21776754

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1379041

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1132745

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