RAGE limits regeneration after massive liver injury by coordinated suppression of TNF-alpha and NF-kappaB - Wikidata - wikimedia - TriplyDB

RAGE limits regeneration after massive liver injury by coordinated suppression of TNF-alpha and NF-kappaB

RAGE limits regeneration after massive liver injury by coordinated suppression of TNF-alpha and NF-kappaB

http://www.wikidata.org/entity/Q41853060

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Contribution of the toxic advanced glycation end-products-receptor axis in nonalcoholic steatohepatitis-related hepatocellular carcinoma Role of receptor for advanced glycation end products (RAGE) in liver disease The NF-κB subunit RelA/p65 is dispensable for successful liver regeneration after partial hepatectomy in mice Soluble RAGE Treatment Delays Progression of Amyotrophic Lateral Sclerosis in SOD1 Mice.Receptor for advanced glycation end products is detrimental during influenza A virus pneumonia.Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE-/- mice RAGE modulates hypoxia/reoxygenation injury in adult murine cardiomyocytes via JNK and GSK-3beta signaling pathways.Ringer's lactate improves liver recovery in a murine model of acetaminophen toxicity.Soluble RAGE: therapy and biomarker in unraveling the RAGE axis in chronic disease and aging High mobility group B1 impairs hepatocyte regeneration in acetaminophen hepatotoxicity Specific activin receptor-like kinase 3 inhibitors enhance liver regeneration Involvement of the TAGE-RAGE system in non-alcoholic steatohepatitis: Novel treatment strategies Expression of RAGE and HMGB1 in thymic epithelial tumors, thymic hyperplasia and regular thymic morphology.Serum levels of sRAGE, the soluble form of receptor for advanced glycation end products, are associated with inflammatory markers in patients with type 2 diabetes Ligands of the receptor for advanced glycation end products, including high-mobility group box 1, limit bacterial dissemination during Escherichia coli peritonitis.Advanced glycation end products and RAGE: a common thread in aging, diabetes, neurodegeneration, and inflammation.Ethyl pyruvate reduces liver injury at early phase but impairs regeneration at late phase in acetaminophen overdose Improved prescription of taohechengqi-tang alleviates D-galactosamine acute liver failure in rats RAGE deficiency improves postinjury sciatic nerve regeneration in type 1 diabetic mice.Soluble receptor for advanced glycation end products and risk of liver cancer.Mechanisms and outcomes of drug- and toxicant-induced liver toxicity in diabetes.Matrix metalloproteinase-9 in the initial injury after hepatectomy in mice.Follicular fluid soluble receptor for advanced glycation endproducts (sRAGE): a potential protective role in polycystic ovary syndrome.Bench-to-bedside review: The inflammation-perpetuating pattern-recognition receptor RAGE as a therapeutic target in sepsis.Prolonged treatment with N-acetylcystine delays liver recovery from acetaminophen hepatotoxicity.IL-1β, RAGE and FABP4: targeting the dynamic trio in metabolic inflammation and related pathologies.Receptor for AGE (RAGE) and its ligands-cast into leading roles in diabetes and the inflammatory response Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation Markedly Increased High-Mobility Group Box 1 Protein in a Patient with Small-for-Size Syndrome.Tanshinone IIA protects rabbits against LPS-induced disseminated intravascular coagulation (DIC).What is critical for liver surgery and partial liver transplantation: size or quality?What is the role of the receptor for advanced glycation end products-ligand axis in liver injury?The role of receptor for advanced glycation endproducts (RAGE) in infection.Targeting the Receptor for Advanced Glycation Endproducts (RAGE): A Medicinal Chemistry Perspective.Maillard reaction in food allergy: Pros and cons.Liver regeneration - mechanisms and models to clinical application.Early Graft Dysfunction in Living Donor Liver Transplantation and the Small for Size Syndrome.Opposing roles of RAGE and Myd88 signaling in extensive liver resection.CCR5 knockout mice with C57BL6 background are resistant to acetaminophen-mediated hepatotoxicity due to decreased macrophages migration into the liver.Carbon monoxide enhances early liver regeneration in mice after hepatectomy.

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RAGE limits regeneration after massive liver injury by coordinated suppression of TNF-alpha and NF-kappaB

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Alan Weinberg

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Ann Marie Schmidt

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Daniel G Jenkins

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Guellue Cataldegirmen

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Hao Dun

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Jay Lefkowitch

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Jean C Emond

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Ling Ling Rong

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Marion A Hofmann

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Nikalesh Ippagunta

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Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

Induction of early-immediate genes by tumer necrosis factor alpha contribute to liver repair following chemical-induced hepatotoxicity

HMG-1 as a late mediator of endotoxin lethality in mice

Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappa B

High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes

N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression

Liver regeneration

Suppression of TNF-alpha-induced apoptosis by NF-kappaB

Nonalcoholic steatohepatitis: a proposal for grading and staging the histological lesions.

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10.1084/JEM.20040934

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201

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8033783

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15699076

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2213026

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