An integrated serotonin and octopamine neuronal circuit directs the release of an endocrine signal to control C. elegans body fat.
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Loss of a neural AMP-activated kinase mimics the effects of elevated serotonin on fat, movement, and hormonal secretionsGain-of-Function Alleles in Caenorhabditis elegans Nuclear Hormone Receptor nhr-49 Are Functionally DistinctPrioritizing Environmental Chemicals for Obesity and Diabetes Outcomes Research: A Screening Approach Using ToxCast™ High-Throughput DataExpression profile of Caenorhabditis elegans mutant for the Werner syndrome gene ortholog reveals the impact of vitamin C on development to increase life span.Lipid droplet protein LID-1 mediates ATGL-1-dependent lipolysis during fasting in Caenorhabditis elegans.Measuring Food Intake and Nutrient Absorption in Caenorhabditis elegans.Multiple excitatory and inhibitory neural signals converge to fine-tune Caenorhabditis elegans feeding to food availability.Pheromone-sensing neurons regulate peripheral lipid metabolism in Caenorhabditis elegansRegulation of body fat in Caenorhabditis elegans.C. elegans Body Cavity Neurons Are Homeostatic Sensors that Integrate Fluctuations in Oxygen Availability and Internal Nutrient ReservesMitochondrial metabolites extend lifespan.Octopamine connects nutrient cues to lipid metabolism upon nutrient deprivation.The Intestinal Copper Exporter CUA-1 Is Required for Systemic Copper Homeostasis in Caenorhabditis elegans.A tachykinin-like neuroendocrine signalling axis couples central serotonin action and nutrient sensing with peripheral lipid metabolism.The ETS-5 transcription factor regulates activity states in Caenorhabditis elegans by controlling satiety.Insights and challenges in using C. elegans for investigation of fat metabolism.Antagonistic Serotonergic and Octopaminergic Neural Circuits Mediate Food-Dependent Locomotory Behavior in Caenorhabditis elegans.Investigating Connections between Metabolism, Longevity, and Behavior in Caenorhabditis elegans.Current advances in the functional studies of fatty acids and fatty acid-derived lipids in C. elegansNuclear receptors: emerging drug targets for parasitic diseases.Neuroendocrine Coordination of Mitochondrial Stress Signaling and Proteostasis.Neuronal CRTC-1 governs systemic mitochondrial metabolism and lifespan via a catecholamine signal.An Aversive Response to Osmotic Upshift in Caenorhabditis elegans.Transcriptional control of satiety in Caenorhabditis elegans.Lipid and Carbohydrate Metabolism in Caenorhabditis elegans.Oxygen-sensing neurons reciprocally regulate peripheral lipid metabolism via neuropeptide signaling in Caenorhabditis elegans.Monoamines differentially modulate neuropeptide release from distinct sites within a single neuron pair.Octopaminergic Signaling Mediates Neural Regulation of Innate Immunity in Caenorhabditis elegansPast experience shapes sexually dimorphic neuronal wiring through monoaminergic signalling
P2860
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P2860
An integrated serotonin and octopamine neuronal circuit directs the release of an endocrine signal to control C. elegans body fat.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
An integrated serotonin and oc ...... o control C. elegans body fat.
@en
type
label
An integrated serotonin and oc ...... o control C. elegans body fat.
@en
prefLabel
An integrated serotonin and oc ...... o control C. elegans body fat.
@en
P2093
P2860
P921
P1433
P1476
An integrated serotonin and oc ...... o control C. elegans body fat.
@en
P2093
Jonathan Stieglitz
Supriya Srinivasan
Tallie Noble
P2860
P304
P356
10.1016/J.CMET.2013.09.007
P577
2013-10-10T00:00:00Z