Mesenchymal cell survival in airway and interstitial pulmonary fibrosis.
about
The effects of carbon nanotubes on lung and dermal cellular behaviorsDispersal state of multiwalled carbon nanotubes elicits profibrogenic cellular responses that correlate with fibrogenesis biomarkers and fibrosis in the murine lungResveratrol inhibits paraquat-induced oxidative stress and fibrogenic response by activating the Nrf2 pathwayGenotoxicity and carcinogenic potential of carbon nanomaterialsAn Allergic Lung Microenvironment Suppresses Carbon Nanotube-Induced Inflammasome Activation via STAT6-Dependent Inhibition of Caspase-1Surface interactions with compartmentalized cellular phosphates explain rare earth oxide nanoparticle hazard and provide opportunities for safer design.Surface charge and cellular processing of covalently functionalized multiwall carbon nanotubes determine pulmonary toxicityUse of a pro-fibrogenic mechanism-based predictive toxicological approach for tiered testing and decision analysis of carbonaceous nanomaterialsDirect isolation of myofibroblasts and fibroblasts from bleomycin-injured lungs reveals their functional similarities and differencesAspect ratio plays a role in the hazard potential of CeO2 nanoparticles in mouse lung and zebrafish gastrointestinal tractImplementation of alternative test strategies for the safety assessment of engineered nanomaterialsDifferences in the Toxicological Potential of 2D versus Aggregated Molybdenum Disulfide in the LungPluronic F108 coating decreases the lung fibrosis potential of multiwall carbon nanotubes by reducing lysosomal injuryAtomic layer deposition coating of carbon nanotubes with aluminum oxide alters pro-fibrogenic cytokine expression by human mononuclear phagocytes in vitro and reduces lung fibrosis in mice in vivoInnate Immune Responses to Nanoparticle Exposure in the LungAtomic layer deposition coating of carbon nanotubes with zinc oxide causes acute phase immune responses in human monocytes in vitro and in mice after pulmonary exposureRepetitive Dosing of Fumed Silica Leads to Profibrogenic Effects through Unique Structure-Activity Relationships and Biopersistence in the LungMultiwalled Carbon Nanotube Functionalization with High Molecular Weight Hyaluronan Significantly Reduces Pulmonary InjurySTAT1-dependent and -independent pulmonary allergic and fibrogenic responses in mice after exposure to tangled versus rod-like multi-walled carbon nanotubesABCG2pos lung mesenchymal stem cells are a novel pericyte subpopulation that contributes to fibrotic remodeling.Mesenchymal-specific deletion of C/EBPβ suppresses pulmonary fibrosis.Nickel nanoparticles enhance platelet-derived growth factor-induced chemokine expression by mesothelial cells via prolonged mitogen-activated protein kinase activationRole of stem/progenitor cells in reparative disorders.The potential for resident lung mesenchymal stem cells to promote functional tissue regeneration: understanding microenvironmental cues.Nickel nanoparticles cause exaggerated lung and airway remodeling in mice lacking the T-box transcription factor, TBX21 (T-bet)Myofibroblast differentiation and survival in fibrotic disease.Emerging in vitro models for safety screening of high-volume production nanomaterials under environmentally relevant exposure conditions.The Genetic Heterogeneity among Different Mouse Strains Impacts the Lung Injury Potential of Multiwalled Carbon Nanotubes.Polyhexamethylene guanidine phosphate aerosol particles induce pulmonary inflammatory and fibrotic responses.Periostin regulates fibrocyte function to promote myofibroblast differentiation and lung fibrosis.Antifibrotic effects of curcumin are associated with overexpression of cathepsins K and L in bleomycin treated mice and human fibroblasts.Immunohistochemical localization of epidermal growth factor system in the lung of the Japanese quail (Coturnix coturnix japonica) during the post-hatching period.Mechanisms of carbon nanotube-induced pulmonary fibrosis: a physicochemical characteristic perspective.Structure Activity Relationships of Engineered Nanomaterials in inducing NLRP3 Inflammasome Activation and Chronic Lung Fibrosis.Fibroblasts Promote Inflammation and Pain via Interleukin-1α-Induction of the Monocyte Chemoattractant CCL2.Loss of lung WWOX expression causes neutrophilic inflammation.Transcriptome analysis reveals differential splicing events in IPF lung tissue.Dasatinib attenuated bleomycin-induced pulmonary fibrosis in mice.Stem cell therapy in chronic obstructive pulmonary disease. How far is it to the clinic?
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P2860
Mesenchymal cell survival in airway and interstitial pulmonary fibrosis.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Mesenchymal cell survival in airway and interstitial pulmonary fibrosis.
@en
type
label
Mesenchymal cell survival in airway and interstitial pulmonary fibrosis.
@en
prefLabel
Mesenchymal cell survival in airway and interstitial pulmonary fibrosis.
@en
P2860
P356
P1476
Mesenchymal cell survival in airway and interstitial pulmonary fibrosis.
@en
P2093
James C Bonner
P2860
P2888
P356
10.1186/1755-1536-3-15
P577
2010-08-25T00:00:00Z
P5875
P6179
1014033101